Interaction of Immunoglobulin E and Cigarette Smoke

Casterline, Charlotte L.

doi:10.1378/chest.84.6.652

Cited by 6 publications

(1 citation statement)

References 9 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…In addition, among nonatopic individuals, bronchial responsiveness occurs more frequently in current smokers as compared with non-or ex-smokers; however, in atopic subjects smoking does not lead to increased prevalence of airway responsiveness [36]. Although the relationship between current smoking and elevated levels of total IgE, as observed in epidemiological studies, is not fully understood [36,37], stimulation of TH2-type (interleukin-4) cytokine production by smoking may represent one mechanistic link [38±42]. Thereby, current smoking may be associated with the unknown serum factor that is linked to total IgE and causes non-speci®c airway responsiveness [7].…”

Section: Discussionmentioning

confidence: 99%

Serum immunoglobulin E levels predict human airway reactivity in vitro

Schmidt

Watson

Ruehlmann

et al. 2000

Clin Experimental Allergy

View full text Add to dashboard Cite

show abstract

Section: Discussionmentioning

confidence: 99%

Serum immunoglobulin E levels predict human airway reactivity in vitro

Schmidt

Watson

Ruehlmann

et al. 2000

Clin Experimental Allergy

View full text Add to dashboard Cite

show abstract

The use of lung function tests in identifying factors that affect lung growth and aging

Buist

Vollmer

1988

Statistics in Medicine

View full text Add to dashboard Cite

Lung function tests are used both clinically, in assessing disease, and epidemiologically, in identifying those factors which influence the growth and aging process of the lungs. The user must beware of several common pitfalls in the use of these tests, however. First, the commonly used tests of lung function can only identify patterns of dysfunction, not specific pathologic processes. Second, these tests are subject to many sources of intra- and inter-subject variability, making it difficult to dissect out the signal (for example, the rate of lung aging in adults) from the noise which may greatly exceed the signal. Finally, the analysis of longitudinal pulmonary function data is complicated by the alinearity of the growth and aging process, missing data, variable follow-up times, information censoring mechanisms and covariate processes, and problems in defining abnormality.

show abstract

Passive smoking and childhood asthma

Willers¹,

Svenonius

Skarping

1991

Allergy

View full text Add to dashboard Cite

Passive exposure to tobacco smoke was assessed in children with asthma (age 3-15) and in referents. There was statistically significantly (P less than 0.0005) higher excretion of the nicotine metabolite, cotinine, in the urine of 49 children with asthma (geometric mean 10 ng/ml) compared with 77 referents (4.8 ng/ml). Maternal smoking was statistically significantly more prevalent among the asthmatics than among the referents (relative risk = RR = 2.6, 95% C1 = 1.2-5.3). In conclusion, the exposure to environmental tobacco smoke in asthmatic children was higher than among healthy children, indicating that passive smoking may be a predisposing and/or aggravating factor for childhood asthma.

show abstract

Interaction of Immunoglobulin E and Cigarette Smoke

Cited by 6 publications

References 9 publications

Serum immunoglobulin E levels predict human airway reactivity in vitro

Serum immunoglobulin E levels predict human airway reactivity in vitro

The use of lung function tests in identifying factors that affect lung growth and aging

Passive smoking and childhood asthma

Contact Info

Product

Resources

About