Inleiding

Feiken, Judith; Jonkers, Roel

doi:10.1007/978-90-313-9060-1_1

Cited by 1 publication

(1 citation statement)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…De Witte et al (2017) reported on a patient with a cerebellar lesion who exhibited apraxia of speech but not ataxic dysarthria. The authors defined apraxia of speech as articulatory groping and segmentation, which they diagnosed using the Diagnostic Instrument Apraxia of Speech (Feiken and Jonkers, 2012). The authors defined ataxic dysarthria as distorted vowels, imprecise consonants, excess and equal stress, and irregular articulatory breakdowns, which they diagnosed using ScreeLing, which is a linguistic screening test that was designed to assess the presence of aphasia (Visch-Brink et al, 2010).…”

Section: Discussionmentioning

confidence: 99%

Aphasia Induced by Infratentorial Ischemic Stroke: Two Case Reports

Inatomi

Nakajima

Yonehara

2021

Cognitive and Behavioral Neurology

View full text Add to dashboard Cite

Aphasia induced by an infratentorial stroke has rarely been reported, and its mechanism has not been fully identified. We evaluated two individuals who had been admitted to Saiseikai Kumamoto Hospital in Kumamoto, Japan, due to acute ischemic stroke in order to determine whether their aphasia was induced by an infratentorial stroke. The first patient, a 59-year-old man with a history of left parietal embolic stroke with very mild sequelae of anomia, developed Wernicke’s aphasia, nonfluent speech, and right limb ataxia as a result of the stroke. The second patient, a 76-year-old woman with a history of chronic renal failure, experienced transcortical sensory aphasia and right one-and-a-half syndrome as a result of the stroke. Both patients’ recent ischemic lesions were limited to the right cerebellar hemisphere and the right medial portion of the midbrain. However, SPECT showed low-uptake lesions in both patients’ left cerebral hemisphere that did not include the recent ischemic lesions but that had spread to an extent that was difficult to be explained by the old or recent ischemic lesions and that might be responsible for their recent aphasia. We believe that the aphasia experienced by these two patients may have been caused by crossed cerebello-cerebral diaschisis.

show abstract

Section: Discussionmentioning

confidence: 99%