1989
DOI: 10.1016/0090-8258(89)90851-2
|View full text |Cite
|
Sign up to set email alerts
|

Inhibition of human ovarian cancer cell proliferation in vitro by neuroendocrine hormones

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2
1
1
1

Citation Types

0
6
0
3

Year Published

1990
1990
2011
2011

Publication Types

Select...
7
1

Relationship

1
7

Authors

Journals

citations
Cited by 24 publications
(12 citation statements)
references
References 9 publications
0
6
0
3
Order By: Relevance
“…Nonetheless, anticancer actions of melatonin against ovarian cancer are inconsistent. Kikuchi et al [43] previously reported that melatonin had no inhibitory effect on the proliferation of ovarian cancer cells compared with other neuroendocrine hormones. In contrast, Bartsch et al [44] observed that melatonin was an inhibitor of ovarian carcinoma in individual cases of patients.…”
Section: Discussionmentioning
confidence: 98%
See 1 more Smart Citation
“…Nonetheless, anticancer actions of melatonin against ovarian cancer are inconsistent. Kikuchi et al [43] previously reported that melatonin had no inhibitory effect on the proliferation of ovarian cancer cells compared with other neuroendocrine hormones. In contrast, Bartsch et al [44] observed that melatonin was an inhibitor of ovarian carcinoma in individual cases of patients.…”
Section: Discussionmentioning
confidence: 98%
“…Kikuchi et al. [43] previously reported that melatonin had no inhibitory effect on the proliferation of ovarian cancer cells compared with other neuroendocrine hormones. In contrast, Bartsch et al.…”
Section: Discussionmentioning
confidence: 99%
“…We have also demonstrated that the KF cell proliferation was dose-dependently inhibited by endorphins and to less extent [Met]enkephalin [7]. Thus, in the present study we attempted to determine effects of opioid peptides on natutal or KF-cell-specific cytotoxicity in spleen cells of intact or tumor-bearing nude mice.…”
Section: Offprint Requests To: Y Kikuchimentioning
confidence: 82%
“…Le traitement par la PGD2 de deux lignées ovariennes tumorales d'origine épithéliale (BG1) ou provenant de la granulosa (Cov434) induit une diminution de leur croissance et une augmentation de leur apoptose, via le récepteur DP1 et l'activation de l'expression de SOX9 ( Figure 3B) [45]. Une telle inhibition des cellules ovariennes KF (dérivées d'un adénocarcinome séreux humain) a été rapportée antérieurement in vitro [46] et in vivo dans des expériences de xénogreffes [47]. Par ailleurs, l'activation de l'expression de la L-PGDS par l'acide rétinoïque est responsable de l'inhibition de croissance des cellules ovariennes tumorales 3AO en culture [48].…”
Section: Revuesunclassified