1991
DOI: 10.1097/00003086-199103000-00021
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Influences of the Protected Passive Mobilization Interval on Flexor Tendon Healing

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Cited by 82 publications
(28 citation statements)
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“…Ferretti et al [17] demonstrated that the application of continuous passive mobilization in a rabbit joint arthrosis model has powerful anti-inflammatory effects, mediated by IL-10 production and the suppression of GAGs degradation. This results in an efficient qualitative and quantitative reorganization of the collagen tissue [18], as well as increased tendon vascularization [19] and metabolic activity of the tenocytes [20,21].…”
Section: Discussionmentioning
confidence: 99%
“…Ferretti et al [17] demonstrated that the application of continuous passive mobilization in a rabbit joint arthrosis model has powerful anti-inflammatory effects, mediated by IL-10 production and the suppression of GAGs degradation. This results in an efficient qualitative and quantitative reorganization of the collagen tissue [18], as well as increased tendon vascularization [19] and metabolic activity of the tenocytes [20,21].…”
Section: Discussionmentioning
confidence: 99%
“…10 We appraised each study and calculated a total MINORS score from a maximum of 16 for noncomparative studies and 24 for comparative studies. Of the studies included, only 2 11,12 were prospective randomized controlled trials. We eval- uated these studies using the Detsky quality score, which is a validated tool to assess the quality of randomized controlled trials.…”
Section: Quality Assessment Of Included Studiesmentioning
confidence: 99%
“…11 Of the observational studies, 27 were low quality, whereas 8 were high quality according to the MINORS score (Appendix A, available on the Journal's Web site at www.jhandsurg.org). There was no significant difference in the proportion of high-quality papers before and after the publication date of January 1, 2000.…”
Section: Quality Assessmentmentioning
confidence: 99%
“…It is generally acknowledged that mobilization early after tendon repair can effectively restore the gliding surface of the injured flexor tendon, improve tendon excursion, limit adhesion formation, and augment strength of the repair site [7,9,10,12]. This theory has been morphologically and histologically supported in animal models [1,8,21].…”
Section: Introductionmentioning
confidence: 99%