2003
DOI: 10.1038/sj.ejcn.1601713
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Influence of renal function on spontaneous dietary intake and on nutritional status of chronic renal insufficiency patients

Abstract: Objective: To analyze the spontaneous food intake and the nutritional parameters of patients with different degrees of chronic renal insufficiency (CRI) at the onset of predialysis treatment. Design: Cross-sectional. Setting: Outpatient Clinic of the Nephrology Division of Federal University of São Paulo, Brazil. Subjects: The analysis involved 487 (187 women and 300 men) patients with moderate to advanced CRI who were evaluated in the first visit to the clinic. Results: Patients were divided according to crea… Show more

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Cited by 51 publications
(35 citation statements)
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“…22,23 Although few studies exist in CKD stages 1-3, a progressive, spontaneous decrease in food intake occurs with greater loss of kidney function, which correlates with accumulation of nitrogen-derived uremic toxins. [24][25][26] The factors influencing food intake involve not only metabolic signals but also anomalies in the digestive system and psychological and acquired aspects, including a desire for pleasure, social behavior, and customs. 27 Anorexia may be mediated by circulating appetite regulators, such as gastric mediators (such as cholecystokinin, 28 peptide YY, 29 ghrelin, 30,31 or obestatin), adipokines (such as leptin 32 and visfatin 33 ), or cytokines (such as tumor necrosis factor [TNF], interleukin [IL]-6, and IL-1b 19,20 ), but these mediators need additional research in the uremic milieu.…”
Section: Undernutrition and Anorexiamentioning
confidence: 99%
“…22,23 Although few studies exist in CKD stages 1-3, a progressive, spontaneous decrease in food intake occurs with greater loss of kidney function, which correlates with accumulation of nitrogen-derived uremic toxins. [24][25][26] The factors influencing food intake involve not only metabolic signals but also anomalies in the digestive system and psychological and acquired aspects, including a desire for pleasure, social behavior, and customs. 27 Anorexia may be mediated by circulating appetite regulators, such as gastric mediators (such as cholecystokinin, 28 peptide YY, 29 ghrelin, 30,31 or obestatin), adipokines (such as leptin 32 and visfatin 33 ), or cytokines (such as tumor necrosis factor [TNF], interleukin [IL]-6, and IL-1b 19,20 ), but these mediators need additional research in the uremic milieu.…”
Section: Undernutrition and Anorexiamentioning
confidence: 99%
“…Chronic kidney disease is often accompanied by reductions in serum albumin and prealbumin and progressive loss of muscle and adipose tissue, likely due to inadequate macronutrient intake, inflammation (1), metabolic acidosis (2,3), reduced physical activity (4), or a combination of these processes (5,6,7). Protein-energy wasting (PEW) generally tends to progress slowly once dialysis is initiated (8,9).…”
Section: Introductionmentioning
confidence: 99%
“…Urinary sodium and urea excretion at the last visit were not considered to avoid an undesirable adjustment for sequelae, 35 related to an anorectic decrease in nutritional intake just before the start of dialysis in patients progressing to ESRD. 42 All analyses were also performed using nonnormalized sodium excretion as an independent variable and the two sodium metrics were compared through Bayesian information criteria. 43 All statistical analyses were performed using R software (version 2.5.1).…”
Section: Statistical Analysesmentioning
confidence: 99%