Influence of non-Newtonian viscosity of blood on microvascular impairment

Abstract

The present research investigated the role of blood viscosity on flow within a microvascular network to identify the conditions of blood flow stagnation. When the yield stress of blood was less than 0.005 Pa, there were no stagnant regions in the microvasculature. However, when the yield stress increased to 0.05 Pa, stagnant or reduced flow areas began to appear, which grew and expanded rapidly with further increase in the yield stress. Thus, the yield stress determined from blood viscosity profile of a patient can be utilized to evaluate the risk of circulatory impairment.

The Interplay of Aging, Aortic Stiffness and Blood Viscosity in Atherogenesis

“…LDL and fibrinogen increase the yield strength of erythrocyte aggregates, i.e. the stress or force needed to permanently deform them [29,30] . This will allow erythrocyte aggregations to enlarge, leading to larger areas of stasis, triggering thrombus formation, a process recently reviewed by Wagner et al [31] .…”

Moment analysis of unsteady bi-component species (drug) transport with coupled chemical reaction in non-Newtonian blood flow

“…Vasu et al [19] also implemented the Eringen micropolar model to compute the timedependent nano-pharmacodynamic transport in a tapered stenotic vessel. Other non-Newtonian models which have been employed to analyse blood flow include Jeffery's viscoelastic model [20], the generalized Oldroyd-B fluid model [21] and the modified Herschel-Bulkley model [18,22].…”