Inferior Oblique Schwannoma: Diagnosis and Management

Colapinto, Patricio; Sheth, Hiten G; Jain, Rajni; Joshi, Naresh; Wong, Tina

doi:10.1080/01676830601174585

Cited by 10 publications

(4 citation statements)

References 5 publications

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“…6 Schwannoma typically grows slowly, with proptosis progressing over the course of years. It is associated with neurofibromatosis type 2, 7 although there were no signs of neurofibromatosis in our patient. When considering schwannoma as a diagnosis, compatible CT features include a well-demarcated hypo-or isodense homogenous lesion with no bony invasion or muscle infiltration and the presence of bony remodeling.…”

Section: T a G G E D H 1 Referencest A G G E D Econtrasting

confidence: 52%

Uncommon presentation of a rapid-onset oculomotor schwannoma in a child

Wesolosky

Campos-Baniak

Sharma

et al. 2020

Canadian Journal of Ophthalmology

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legally blind. 3 Nystagmus and abnormal smooth pursuit were also common. As in our case, a subset of patients exhibited cortico-spinal tract signs. The diagnosis of Boucher-Neuhauser syndrome is potentially challenging as there is considerable variability in clinical presentation. Fundus abnormalities may be subtle with relatively preserved central acuity, 4 or more there may be extensive chorioretinal atrophy with severe central vision loss. 5 In the more frequent cases where vision is significantly compromised, decreased central acuity is accompanied by abnormal colour vision and ring scotomas on visual field testing. Electroretinogram typically shows decreases in both photopic and scotopic responses. 5,6 Anterior segment examination is characteristically unremarkable. In this case, early nyctalopia and peripheral field loss was suggestive of RP; however, the fundus did not show peripheral pigment clumping/bone spicule pigmentation, waxy disc pallor, or marked retinal vessel attenuation, and posterior subcapsular cataract, also commonly seen in RP, was not present. The associated findings of cerebellar ataxia, spasticity, and delayed puberty pointed to the correct diagnosis.

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Section: T a G G E D H 1 Referencest A G G E D Econtrasting

confidence: 52%

Uncommon presentation of a rapid-onset oculomotor schwannoma in a child

Wesolosky

Campos-Baniak

Sharma

et al. 2020

Canadian Journal of Ophthalmology

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“…Recently, several authors have reported cases of endoscopic coblation-assisted surgery in juvenile angiofibroma; however, clinical studies considering endoscopic harmonic scalpel-assisted surgery for vascular tumors of the anterior skull base are still lacking. 5 In that regard, we have been using the harmonic scalpel in our endoscopic skull base practice since 2008, with promising results. This has reduced the duration of surgical procedures by significantly reducing the amount of intraoperative bleeding, thus allowing for a more straightforward procedure and facilitation of dissection of tissues and planes.…”

Section: Discussionmentioning

confidence: 99%

“…Presenting symptoms of this pathology are nonspecific and depend greatly on the localization of the tumor within the orbit, but nonpulsatile ocular proptosis and displacement of the globe are frequent. Schwannomas are encapsulated, meaning they do not infiltrate adjacent structures 5. Any optic nerve or extraocular muscle involvement is therefore because of the direct compression of the structure or its vasculature 1,6.…”

Section: Discussionmentioning

confidence: 99%

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Orbital Schwannoma With Frontal Bone Lysis

Farag

Daigle

2023

Journal of Craniofacial Surgery

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Orbital schwannoma is an exceptionally rare cause of ptosis. Diagnosis may be elusive given its slow rate of growth and its various presentations depending on localization. Herein, we report the case of a 50-year-old male who presented to our clinic with a complaint of unilateral, recurrent ptosis of the left eye. He underwent levator palpebrae resection, which was unsuccessful at improving his ptosis. He later represented with acute-onset diplopia for which magnetic resonance imaging was obtained. Magnetic resonance imaging showed a lesion in the superior orbit with secondary bony dehiscence of the orbital roof. Through a vertical lid-split incision, the lesion was removed, and the frontal lobe was observed protruding through the defect in the orbital roof. This case highlights the importance of diagnostic skepticism in the face of recurrent ptosis and emphasizes the utility of the vertical lid-split approach for anterior lesions of the superior orbit.

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