“…Several theories have been suggested, includ ing damage to the juxtaglomerular apparatus [1,2,7], impaired sympathetic nervous system stimulation [1,2,24], suppression by volume expansion [1,2,6], impaired conversion of 'prorenin' to renin [1,2,[8][9][10], and altera tions in prostaglandin synthesis [1,2,9,11,14,15]. We examined the last two mechanisms in the present study.…”