Abstract:Pacemaker implantation is considered as a standard procedure for treatment of symptomatic bradycardia in both dogs and cats. Advanced second-degree and third-degree atrioventricular blocks, sick sinus syndrome, persistent atrial standstill, and vasovagal syncope are the most common rhythm disturbances that require pacing to either alleviate clinical signs or prolong survival. Most pacemakers are implanted transvenously, using endocardial leads, but rarely epicardial leads may be necessary. To decide whether a … Show more
“…3AVB is classified as congenital or acquired, and can be transient, intermittent or permanent. It results from an interruption of the electrical impulse between atria and ventricles, due to an anatomical or functional impairment in the AV conduction system (Santilli et al, 2019). This case arrhythmia was classified as transient since it was spontaneously reverted to sinus rhythm after the OSH.…”
Section: Discussionmentioning
confidence: 99%
“…Several primary or secondary disorders of the AV conduction system are capable of inducing 3AVB, but usually, the cause is unknown (Willis, 2018). However, many underlying causes for 3AVB in canines have been cited including degeneration and fibrosis of the AV conduction system (Kaneshige et al, 2007), isolated congenital AV block, inherited heart defects, adrenal insufficiency (Patterson et al, 1961), myocarditis, cardiomyopathy, infections, myocardial infarction, hyper-hypokalemia, drugs intoxication (digoxin and calcium channel blockers), cardiac neoplasia, immune-mediated and inflammatory diseases (myasthenia gravis, lupus erythematosus), trauma, inflammation and systemic disorders (Willis, 2018;Santilli et al, 2019) and pancreatitis (Oron et al, 2015).…”
“…3AVB is classified as congenital or acquired, and can be transient, intermittent or permanent. It results from an interruption of the electrical impulse between atria and ventricles, due to an anatomical or functional impairment in the AV conduction system (Santilli et al, 2019). This case arrhythmia was classified as transient since it was spontaneously reverted to sinus rhythm after the OSH.…”
Section: Discussionmentioning
confidence: 99%
“…Several primary or secondary disorders of the AV conduction system are capable of inducing 3AVB, but usually, the cause is unknown (Willis, 2018). However, many underlying causes for 3AVB in canines have been cited including degeneration and fibrosis of the AV conduction system (Kaneshige et al, 2007), isolated congenital AV block, inherited heart defects, adrenal insufficiency (Patterson et al, 1961), myocarditis, cardiomyopathy, infections, myocardial infarction, hyper-hypokalemia, drugs intoxication (digoxin and calcium channel blockers), cardiac neoplasia, immune-mediated and inflammatory diseases (myasthenia gravis, lupus erythematosus), trauma, inflammation and systemic disorders (Willis, 2018;Santilli et al, 2019) and pancreatitis (Oron et al, 2015).…”
“…Cardiac pacing is an effective therapy for various arrhythmias in humans and a therapeutic implantation of a cardiac pacemaker has also been described in a horse [ 1 , 2 , 3 , 4 ] and a donkey [ 5 , 6 ]. Indications for pacemaker implantation are symptomatic bradycardic arrhythmias such as third-degree AV block, high-grade second-degree AV block, sick sinus syndrome or persistent atrial standstill [ 1 , 3 , 5 , 6 , 7 , 8 , 9 , 10 ]. Transvenous pacemaker implantation is a relatively safe and simple procedure.…”
A five-month-old African jenny was presented with a history of exercise intolerance and syncopal episodes. Severe bradycardic arrhythmia due to a high-grade second-degree atrioventricular (AV) block with progression to complete AV block was diagnosed. The jenny underwent a transvenous single-chamber pacemaker implantation. The implantation procedure was performed in a lateral recumbency and the ventricular lead was inserted through the jugular vein. Positioning of the lead was guided by echocardiography. The pacemaker was programmed to VVI mode with a minimal ventricular rate of 40 pulses per minute, a pulse amplitude of 2.4 V, a pulse width of 0.5 ms and sensing amplitude of 2.5 mV. Short-term complications associated with the procedure included lead dislodgement and pacemaker pocket infection. The long-term outcome was satisfactory; the jenny showed improvement in heart function and quality of life after pacemaker implantation. The pulse generator replacement was performed twice (at nine-year intervals) and the intervention was always associated with a local inflammatory reaction around the pacing device. Cardiac examination 18 years after pacemaker implantation revealed no morphological changes in the heart; the electrode lead was still in the correct position and successful pacing and sensing of the ventricle were obtained. Regular follow-up checks are important to evaluate pacemaker function.
“…Permanent pacemaker implantation is indicated in dogs with symptomatic bradycardias including high-grade second degree and third-degree atrioventricular (AV) block, sick sinus syndrome and sinus arrest. 1 Cardiac arrest during implantation of permanent pacemakers is an occasionally reported complication of the surgical procedure itself, occurring consequently to ventricular fibrillation. 2 3 Often, exacerbation of bradycardias may be detected following induction of general anaesthesia in these dogs, with one retrospective study identifying a requirement for temporary transcutaneous pacing (TCP) in 27 out of 77 cases.…”
Third-degree atrioventricular block is a haemodynamically unstable bradycardia frequently resulting in signs of lethargy, weakness and collapse. In this reported case, a four year-four month-old male neutered Cavalier King Charles spaniel diagnosed with third-degree atrioventricular block was referred for transvenous permanent pacemaker implantation. During induction of general anaesthesia, the dog suffered cardiac arrest consistent with ventricular standstill, as indicated by cessation of ventricular electrical activity on the ECG monitor and the absence of a peripheral pulse. The prior placement of transthoracic pacing pads under sedation allowed for rapid commencement of temporary transcutaneous pacing and proved effective in achieving ventricular capture with re-establishment of cardiac output. The subsequent general anaesthesia for implantation of a permanent pacemaker was uneventful. This report considers the possible causes of ventricular escape rhythm suppression and highlights the importance of ensuring availability of a temporary pacing method from the outset when anaesthetising animals with unstable and symptomatic bradycardias.
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