Increased Macrophages, High Serum M‐csf and Low Serum Cholesterol in Myelodysplasia and Kawasaki Disease

Abstract

Fig 1.Karotype of the bone marrow cells by the Giemsa banding technique: 48, XX, 1, add(1)(q21), der(1;8)(q10;q10), del(3)(q26), t(11;14)(q13;q32), 16, 18. Arrowheads indicate the der(11)t(11;14) and the der(14)t(11;14).

Spontaneous bilateral renal aneurysm rupture secondary to Polyarteritis Nodosa in a patient with chronic myelomonocytic leukaemia: A case report study

“…Despite the involvement of B and T cells, macrophage clearance of antigens may be greatly reduced, leading to enhanced levels of circulating immune complexes [5] . In CMML, a high number of circulating monocytes might also contribute to vessel inflammation [6] . By this case report, however, we did not find any explanatory pathophysiological mechanism; the association between CMML and PAN needs further investigation.…”

Systemic vasculitis with bilateral perirenal haemorrhage in chronic myelomonocytic leukaemia

“…Besides the involvement of T or B cells, macrophage clearance of antigens may be greatly reduced, leading to enhanced levels of circulating immune complexes. In chronic myelomonocytic leukaemia, a high number of circulating monocytes might also contribute to vessel inflammation 14. Interestingly, numerous and complex karyotype abnormalities in the myelodysplastic syndrome seem to be associated with extrahaematological manifestations and carry a worse prognosis 15…”