Incontinence and pelvic organ prolapse in parous/nulliparous pairs of identical twins

Buchsbaum, Gunhilde M.; Duecy, Erin E.

doi:10.1002/nau.20555

Cited by 19 publications

(13 citation statements)

References 13 publications

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“…Evidence in the literature supports an association between paediatric and adult lower urinary tract dysfunction. Genetic as well as environmental factors have been proposed to explain this .…”

Section: Discussionmentioning

confidence: 99%

Urological dysfunction in young women: an inheritance of childhood?

et al. 2017

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Section: Discussionmentioning

confidence: 99%

Urological dysfunction in young women: an inheritance of childhood?

et al. 2017

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“…It has been reported that about a third of women have UI after childbirth [Hallock and Handa, ]. In addition to parity, a positive family history of prolapse increases a woman's risk of prolapse, even among nulliparous women [Buchsbaum et al, ; Buchsbaum and Duecy, ].…”

Section: Clinical Descriptionmentioning

confidence: 99%

Hypermobile Ehlers–Danlos syndrome (a.k.a. Ehlers–Danlos syndrome Type III and Ehlers–Danlos syndrome hypermobility type): Clinical description and natural history

Tinkle

Castori²,

Berglund

et al. 2017

American J of Med Genetics Pt C

357

348

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The hypermobile type of Ehlers-Danlos syndrome (hEDS) is likely the most common hereditary disorder of connective tissue. It has been described largely in those with musculoskeletal complaints including joint hypermobility, joint subluxations/dislocations, as well as skin and soft tissue manifestations. Many patients report activity-related pain and some go on to have daily pain. Two undifferentiated syndromes have been used to describe these manifestations-joint hypermobility syndrome and hEDS. Both are clinical diagnoses in the absence of other causation. Current medical literature further complicates differentiation and describes multiple associated symptoms and disorders. The current EDS nosology combines these two entities into the hypermobile type of EDS. Herein, we review and summarize the literature as a better clinical description of this type of connective tissue disorder.

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“…In relation to collagen, joint hypermobility and various collagen diseases such as Ehlers-Danlos or Marfan syndrome are linked with POP [16]. All these factors are indicative of the systemic manifestation of collagen abnormalities and suggest a role for congenital predisposition in POP etiology [17,18]. POP is not a condition caused by pregnancy alone; it could be a clinical manifestation of a preexisting, undiagnosed condition or a congenital predisposition triggered by pregnancy and delivery.…”

Section: Discussionmentioning

confidence: 99%

Is Procidentia a Distinct Disease? Comparison of Risk Factors

Aydın¹

2016

Ann Clin Anal Med

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ÖzetAmaç: Total prolapsus, diğer bir ifadeyle prosidentia vajinanın tümünün dışa dönmesi olarak tanımlanır. Yaş, gebelik doğum, pelvik cerrahi öyküsü ve kabız-lık, kronik pulmoner hastalıklar, obezite gibi karın içi basıncı artıran durumlar pelvic organ prolapsusu için risk faktörleridir. Bu çalışma anterior,posterior, apikal ve total vaginal duvar prolapsusu insidanslarını, kompartmanlar arası, total ve subtotal prolasus arasındaki risk faktörleri açısından farklılıkla-rı belirlemektir. Gereç ve Yöntem: Bu retrospektif çalışma prolapsus cerrahisi geçiren kadınları kapsar. Total ve subtotal prolapsusu olanlar parite, yaş, vücut kitle indeksi, menapoz ve laboratuar verileri gibi potansiyel risk faktör-leri açısından tek yönlü varyans analizi, Ki kare ve Fisher testi kullanılarak kı-yaslandı. Bulgular: Prolasus cerrahisi geçiren 179 kadın arasında 29 kadın total uterovajinal prolapsus olarak değerlendirildi. Subtotal prolapsusu olan kadınlar (29.4±3.7) anlamlı olarak total prolapsusu olanlardan (26.1±3.3) daha şişmandı (p<0.001). Yaş, parite, histerektomi öyküsü ve menapoz durumu açı-sından fark yoktu. Anterior vajinal duvar defekti olanlarda multipartite oranı diğerlerine kıyasla daha azdı (52,4 vs 70.5-88,.9%, p=0.03). Tartışma: Biz total uterin veya vajinal kubbe prolasusun farklı bir durum olduğunu ve yaş ve doğum gibi bilinen risk faktörlerinden faklı bazı spesifik veya belirlenmemiş faktörlele ilişkili olabileceğini varsayımında bulunuyoruz. Total prolasusu olan kadınlar daha inceydi ve anterior vajinalduvar defektleri multipartite ile daha az ilişkiliydi. Bu bulgular ışığında gelecekte yapılacak çalışmalar ile total uterovajinal prolapsusun patofizyolojisini açıklamayı planlayan çalış-malar yapılmalıdır. Anahtar KelimelerPelvik Organ Prolapsusu; Prosidentia; Total Uterovajinal Prolapsus Abstract Aim: Total prolapse-in other words, procidentia-refers to the complete eversion of the total length of the vagina. Age, pregnancy, delivery, previous pelvic surgery, and conditions that increase intra-abdominal pressure, such as chronic pulmonary disease, constipation, and obesity are risk factors for pelvic organ prolapse. This study aims to assess the incidence of anterior, posterior, apical, and total prolapse and to determine differences in the risk factors for different vaginal compartment prolapse, total prolapse, and subtotal prolapse. Material and Method: This is a retrospective study of patients who underwent prolapse surgery. Comparison of possible risk factors such as parity, age, body mass index, menopause, and preoperative laboratory results were done between women with total and subtotal uterovaginal prolapse by using one way analysis of variance, chi-square, and Fisher tests. Results: A total of 29 women had total uterovaginal prolapse among 179 women who underwent prolapse surgery. Women with subtotal prolapse (29.4±3.7) were significantly more obese than women with total prolapse (26.1±3.3), (p<0.001). Age, parity, hysterectomy history, and menopause status were similar. Multiparity was less in wome...

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Incontinence and pelvic organ prolapse in parous/nulliparous pairs of identical twins

Cited by 19 publications

References 13 publications

Urological dysfunction in young women: an inheritance of childhood?

Urological dysfunction in young women: an inheritance of childhood?

Hypermobile Ehlers–Danlos syndrome (a.k.a. Ehlers–Danlos syndrome Type III and Ehlers–Danlos syndrome hypermobility type): Clinical description and natural history

Is Procidentia a Distinct Disease? Comparison of Risk Factors

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