Inclusive. pi. /sup + -/, K/sup + -/, and p, anti p production in e/sup +/e/sup -/ annihilation at. sqrt. s = 29 GeV

Cowan, G.

doi:10.2172/5188194

Cited by 8 publications

(16 citation statements)

References 16 publications

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“…Consistent with research reviewed earlier (Cowan et al, 1993;Lytton & Romney, 1991;Youniss & Smollar, 1985), we found few differences between daughters and sons for the influence of mothers. Whereas mothers significantly affected their sons but not their daughters by promoting prosocial beliefs (and subsequently, behavior), mothers' effects on beliefs were of roughly the same order of magnitude for their daughters and sons.…”

Section: Discussionsupporting

confidence: 91%

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Unique Influence of Mothers and Fathers on Their Children's Antisocial Behavior

Kosterman

Haggerty

Spoth

et al. 2004

J of Marriage and Family

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The social development model (Catalano & Hawkins, 1996) was adapted to examine the unique influence of mothers and fathers on their children's antisocial behavior. Analyses examined 325 families with sixth‐grade children. Structural equation modeling was used to assess unique influences of constructs specific to mothers or fathers. Multiple‐group comparisons were conducted to identify differences in the relationships between constructs for daughters versus sons. Results suggested that, although the relationships were often similar for both parents and for both daughters and sons, mothers and fathers uniquely influenced their child's antisocial behavior depending on the child's gender. Overall, cross‐gender influence appeared to be particularly important for fathers’ control of their daughters’ antisocial behavior. Implications for the prevention of antisocial behavior are discussed.

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Section: Discussionsupporting

confidence: 91%

“…Youniss and Smollar describe the father-daughter relationship as a ''nonrelation' ' (p. 51). Other studies appear to concur that the differential socialization of daughters and sons occurs primarily with fathers (Cowan et al, 1993;Lytton & Romney, 1991).…”

Section: Differential Influence On Daughters and Sonsmentioning

confidence: 77%

Unique Influence of Mothers and Fathers on Their Children's Antisocial Behavior

Kosterman

Haggerty

Spoth

et al. 2004

J of Marriage and Family

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“…Phenotype characterization of RJ2. In previous reports, HLA class I-transfected L cells were sufficient for stimulation of some alloreactive Tcell clones [37], but not others [38], depending apparently on the fine specificity of the clones and their sensitivity to the cellular background on which the allogeneic molecules are expressed In conclusion, the capacity to stimulate Tcell alloresponses was not a simple function of class I1 density, expression of known accessory molecules or invariant chain, the ability to process antigens or the presence of human peptides. One must, therefore, assume that the additional requirement is either for further, undefined, accessory molecules or specific cytokines [27].…”

Section: Discussionmentioning

confidence: 95%

“…Alloreactivity, manifested clinically as graft rejection or GVH disease, represents the cross-reactive recognition of non-self MHC products by a TcR which is specific for In previous reports, HLA class I-transfected L cells were sufficient for stimulation of some alloreactive Tcell clones [37], but not others [38], depending apparently on the fine specificity of the clones and their sensitivity to the cellular background on which the allogeneic molecules are expressed [39]. Our own studies have shown that HLA class 11-transfected L cells stimulated MLR-like responses in PBMC, provided that sufficiently high class I1 expression is achieved.…”

Section: Discussionmentioning

confidence: 99%

Allorecognition of HLA‐DR and ‐DQ transfectants by human CD45RA and CD45R0 CD4 T cells: Repertoire analysis and activation requirements

Merkenschlager

Ikeda²,

Wilkinson³

et al. 1991

Eur J Immunol

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We have investigated the requirements for allogeneic stimulation of human CD4 T cells using HLA class II products expressed on various cellular backgrounds. Human (class II-negative RJ2.2.5 mutant) B cell lines transfected with HLA-DR or -DQ cDNA clones were efficient stimulators for highly purified CD4 T cells. HLA-DR-transfected mouse L cells or IFN-gamma-induced human fibroblasts, although able to function as accessory cells for T cell responses to the mitogen PHA, failed to stimulate strong T cell alloresponses. On the basis of these observations, we have employed class II transfectants to address the following questions: (a) do CD45RA and CD45R0 subpopulations differ in their allogeneic activation requirements, (b) are these subpopulations skewed in their recognition of HLA-DQ vs. HLA-DR in a manner which might support the concept that CD45RA T cells are involved in HLA-DQ-restricted suppressor inducer functions and (c) by using transfectants expressing individual HLA-DR or -DQ heterodimers in combination with limiting dilution analysis, can one for the first time obtain estimates of precursor frequencies for allogeneic cells recognizing each of these class II isotypes? Our results show that CD45RA and CD45R0 T cells respond comparably to optimal numbers of stimulator cells. However, when CD45RA and CD45R0 T cell populations depleted of endogenous accessory cells were cultured with limiting numbers of stimulator cells, CD45R0 cells generally responded more strongly, consistent with the elevated levels of various adhesion molecules known to be expressed by this population. Further, we found a similar representation of responses to HLA-DR and -DQ antigens among populations expressing CD45RA and CD45R0 isoforms. Finally, the precursor frequencies of allogeneic CD4 T cells responding to particular HLA-DR alleles were higher than to -DQ, but only by a factor of about 1.6, indicating that HLA-DQ recognition may occur more frequently than implied from previous antibody blocking studies.

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“…The ability of Tax protein to induce cell growth and influence transcription regulation is consistent with its role as an extracellular cytokine. In addition, treatment of extracellular Tax protein results in the induction of TNFa gene expression in human neuronal cells (53). Such Tax protein-induced lymphocyte proliferation is also associated with activation of endogenous IL-2R a-chain expression (52).…”

Section: Discussionmentioning

confidence: 99%

Extracellular human T cell leukemia virus type I tax protein stimulates the proliferation of human synovial cells

Aono

Fujisawa

Hasunuma

et al. 1998

Arthritis & Rheumatism

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Inclusive. pi. /sup + -/, K/sup + -/, and p, anti p production in e/sup +/e/sup -/ annihilation at. sqrt. s = 29 GeV

Abstract: and a dE/dx resolution of typically 3.5% allow the measurements to be extended to higher z than previously reported.

Cited by 8 publications

References 16 publications

Unique Influence of Mothers and Fathers on Their Children's Antisocial Behavior

Unique Influence of Mothers and Fathers on Their Children's Antisocial Behavior

Allorecognition of HLA‐DR and ‐DQ transfectants by human CD45RA and CD45R0 CD4 T cells: Repertoire analysis and activation requirements

Extracellular human T cell leukemia virus type I tax protein stimulates the proliferation of human synovial cells

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