Immunology Research

Heimberger, Amy B.; Wu, A.; Wang, Y.; Kong, L.-Y.; Doucette, T.; Wei, J.; Priebe, W.; Sawaya, R.; Yang, J.; Rao, G.; Heimberger, A.; Shimato, S.; Meier, L. M.; Castelli, M.; Canoll, P.; Asslaber, M.; Bruce, J. N.; Anderson, D. E.; Anderson, R. C.; Mahlum, E. W.; Jenkins, R. B.; Kohanbash, G.; Mintz, A. H.; McKaveney, K.; McDonald, H. A.; Ohlfest, J. R.; Okada, H.; Fujita, M.; Zhang, L.; Liu, W.; Alizadeh, D.; Zhao, D.; Farrukh, O.; Badie, B.; Raychaudhuri, B.; Pellegatta, S.; Cantini, G.; Pisati, F.; Cuppini, L.; Finocchiaro, G.; Albesiano, E.; Han, J. E.; See, A.; Jackson, C.; Lim, M.; Nag, K.; White, J.; Sippel, T.; Klaassen, M.; Tsvankin, V.; Waziri, A.; Kupsky, W. J.; Alkonyi, B.; Sood, S.; Zitron, I. M.; Juhasz, C.; Mittal, S.

doi:10.1093/neuonc/noq116.s4

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2014

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“…Tumour cells actively secrete factors that skew the phenotype of GAMs to a M2 phenotype, including expression of GM-CSF [79], CSF-1 [75,92], and S100B [93]. Immunosuppressive factors including IL-4, IL-6, IL-10, MIF, TGF-β, and PGE-2 are secreted by glioma cells, and are also known to drive GAM differentiation towards an M2 phenotype rather than an M1 phenotype [47].…”

Section: Recruitment and M2 Phenotype Polarization Of Gams By Glioma mentioning

confidence: 99%