Immediate Primary Transcatheter Closure of Left Ventricular Free Wall Rupture after Myocardial Infarction

Vatan, Bülent; Akdemir, Ramazan; Çakar, Mehmet Akif; Gunduz, Hüseyin; Varım, Ceyhun

doi:10.1159/000348420

Cited by 5 publications

(4 citation statements)

References 8 publications

(4 reference statements)

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“…Additionally, CFWR may occur as a complication of cardiac-based procedure or trauma [4,12]. Significant advancements and increasing use of cardiac-based procedure, such as cardiac catheterization, patent foramen ovale closure, left atrial appendage closure, defibrillator placement, and pericardiocentesis has resulted in an increased incidence of CFWR [4,[13][14][15][16][17][18][19][20][21][22][23][24][25].…”

Section: Discussionmentioning

confidence: 99%

Employing Amplatzer Occluder® in Cardiac Free Wall Rupture Repair: A Scoping Study

Kariyanna¹,

Tadayoni²,

Jayarangaiah³

et al. 2020

AJMCR

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Cardiac free wall rupture (CFWR) is an uncommon complication of myocardial infarction, cardiacbased procedures, and blunt chest trauma. Cardiac tamponade and shock which occurs as a result of CFWR results in a high mortality rate. Despite the high mortality rate, there is a window of opportunity for intervention in selected patients with acute or subacute free wall rupture. Hence, prompt diagnosis and intervention are key to prevent cardiac tamponade and death. Even though emergency surgical repair is the standard treatment for the CWFR, the catheter-based procedure has provided an alternative treatment option, especially, in the high-risk surgical patients. For instance, Amplatzer occluder® (AO), a device which is used in repairing congenital septal wall defect, is being used as an alternative method of treatment in CFWR. In this systemic review, we assessed the 19 cases of CFWR occurring after invasive cardiac procedures who underwent repair with the utilization of AO®. The study shows that the successful rate of percutaneous closure of CFWR was 84.3% (16/19) with a mortality rate of 15.7% (3/19) in this cohort. Therefore, the in-hospital mortality rate of CFWR closure is comparable with the average in-house mortality rate of emergency surgical repair which is 14%. Furthermore, we found that AO® placement technique has a lower mortality rate compared to the other less-invasive methods such as percutaneous intrapericardial fibrin-glue injection which has a mortality rate of 25%. In conclusion, employing AO® in CFWR repair not only serves as the treatment of choice in the high-risk surgical candidates but could also be applied as an alternative method in the general population. However, further studies are required to assess the outcome and mortality rate of using AO® in CFWR to provide us with a more consistent and accurate data.

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Section: Discussionmentioning

confidence: 99%

Employing Amplatzer Occluder® in Cardiac Free Wall Rupture Repair: A Scoping Study

Kariyanna¹,

Tadayoni²,

Jayarangaiah³

et al. 2020

AJMCR

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“…Left ventricular free wall rupture (LVFWR) occurs in about 2-4% of all patients with acute myocardial infarction (AMI) and may be accompanied by an unpredictable risk of sudden cardiac death (1)(2)(3). Most patients have a very short interval from disease onset to death, with little chance for treatment.…”

Section: Introductionmentioning

confidence: 99%

Transcatheter closure for the treatment of pseudoventricular aneurysm after acute myocardial infarction: a case report

et al. 2020

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Left ventricular free wall rupture (LVFWR) is a rare but lethal complication of acute myocardial infarction (AMI). Urgent surgery is essential but associated with high postoperative mortality. Even worse, LVFWR patients may experience sudden death without a chance for surgery. In this article, we report our successful transcatheter closure of a patient with the most extensive pseudoventricular aneurysm after AMI reported thus far. Cardiac magnetic resonance imaging (MRI) revealed a giant pseudoventricular aneurysm located in the inferior and lateral walls of the left ventricle; the rupture diameter was 28 mm, and the maximum tumor diameter was 90.2 mm. We used transcarotid approach (TCA) and atrial septal defect closure umbrella to complete the operation, which solved the lack of special interventional instruments to treat pseudoventricular aneurysm after AMI. In addition, we still needed to treat liver and kidney failure caused by hemolysis after operation, and undergone strict follow-up. In conclusion, transcatheter closure is practical and feasible for the treatment of pseudoventricular aneurysm after AMI, although hemolysis and decline of cardiac pumping function after the successful interventional treatment deserve special attention.Future multicenter studies are required to identify patients best suited for interventional treatment timing.And further developments in devices and delivery techniques are required in order to optimize interventional outcomes.

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“…Although cases of left ventricular free wall rupture in avian species have not been documented previously, to our knowledge, multiple cases in the human medical literature describe rupture of the left and right ventricular free walls, as well as ventricular septal rupture, as complications of myocardial infarction. 20,22 Turkeys can be afflicted by several cardiovascular diseases including spontaneous aortic rupture; however, such diseases are more common in commercial rather than backyard turkeys given the differences in management and environment. Spontaneous aortic rupture occurs typically in 12-16-wk-old heavy and fast-growing male turkeys.…”

mentioning

confidence: 99%

mentioning

confidence: 99%

Fowl cholera and acute heart rupture in a backyard turkey

et al. 2019

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Fowl cholera is caused by the gram-negative non-motile bacterium Pasteurella multocida. P. multocida subspecies multocida is the main cause of fowl cholera, although occasional outbreaks are caused by subspecies septica or gallicida. Fowl cholera occurs in most avian species and is contagious; turkeys are very susceptible, with infections frequently resulting in high mortality in a flock. 7 Fowl cholera is frequently seen as an acute septicemic disease with high mortality and gross anatomic lesions resulting from vascular disturbances. Other domesticated farm animals are carriers and can shed P. multocida; strains vary in virulence for avian species. 10 Once infection occurs, birds may succumb in the acute septicemic phase, may survive the acute phase and succumb in the days following, may become chronically infected, or may ultimately recover. 7 P. multocida can enter the host through the mucous membranes of the upper respiratory tract, conjunctiva, and cutaneous wounds. 7 Stressors, such as changes in environment, nutrition, and injury, can affect the course of the disease. Chronically infected carrier and convalescent birds are a major source of infection because carrier birds shed the bacteria for life. 7 Chronic conditions can occur in which localized infections predominate. 7,9,14 P. multocida can survive in the environment for up to 2 wk and in dried blood for up to 8 d. 7 If separated from organic material, P. multocida is destroyed easily by common disinfectants, sunlight, drying, and at temperatures >56°C. 7 3850V DIXXX10.1177/1040638718823850Fowl cholera and heart rupture in a turkeyBlakey et al.

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Immediate Primary Transcatheter Closure of Left Ventricular Free Wall Rupture after Myocardial Infarction

Cited by 5 publications

References 8 publications

Employing Amplatzer Occluder® in Cardiac Free Wall Rupture Repair: A Scoping Study

Employing Amplatzer Occluder® in Cardiac Free Wall Rupture Repair: A Scoping Study

Transcatheter closure for the treatment of pseudoventricular aneurysm after acute myocardial infarction: a case report

Fowl cholera and acute heart rupture in a backyard turkey

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