Imidacloprid-induced lung injury in mice: Activation of the PI3K/AKT/NF-κB signaling pathway via TLR4 receptor engagement

Xie, Wen; Chen, Canrong; Li, Heming; Tu, Yuxin; Zhong, Yanhui; Lin, Zian; Cai, Zongwei

doi:10.1016/j.scitotenv.2024.172910

Science of The Total Environment

2024

DOI: 10.1016/j.scitotenv.2024.172910

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Imidacloprid-induced lung injury in mice: Activation of the PI3K/AKT/NF-κB signaling pathway via TLR4 receptor engagement

Wen Xie,

Canrong Chen,

Heming Li

et al.

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Cited by 2 publications

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Imidacloprid unique and repeated treatment produces cholinergic transmission disruption and apoptotic cell death in SN56 cells

Moyano,

Flores,

San Juan

et al. 2024

Food and Chemical Toxicology

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Imidacloprid unique and repeated treatment produces cholinergic transmission disruption and apoptotic cell death in SN56 cells

Moyano,

Flores,

San Juan

et al. 2024

Food and Chemical Toxicology

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Light-powered biodegradation of Imidacloprid by Scenedesmus sp. TXH202001: Assessing complete removal, metabolic pathways, and toxicity verification

Cheng,

Wang,

et al. 2024

Journal of Hazardous Materials

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Imidacloprid-induced lung injury in mice: Activation of the PI3K/AKT/NF-κB signaling pathway via TLR4 receptor engagement

Cited by 2 publications

References 66 publications

Imidacloprid unique and repeated treatment produces cholinergic transmission disruption and apoptotic cell death in SN56 cells

Imidacloprid unique and repeated treatment produces cholinergic transmission disruption and apoptotic cell death in SN56 cells

Light-powered biodegradation of Imidacloprid by Scenedesmus sp. TXH202001: Assessing complete removal, metabolic pathways, and toxicity verification

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