Imaging evaluation of cranial nerves 3, 4, and 6

Eisenkraft, Barbara; Ortiz, A. Orlando

doi:10.1016/s0887-2171(01)90003-4

Cited by 15 publications

(8 citation statements)

References 46 publications

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“…Il prend naissance entre les artères cérébelleuse supérieure et cérébrale postérieure, puis se dirige perpendiculairement au tronc basilaire et parallèlement à la communicante posté-rieure. Considérant ses rapports anatomiques, les contacts vasculaires avec le nerf oculomoteur commun ne sont pas rares [1,3,4]. Cependant, les observations de paralysie du 3 e nerf crânien dues à un conflit vasculo-nerveux sont exceptionnelles.…”

Section: Discussionunclassified

Paralysie brutale du nerf oculomoteur commun révélant un conflit vasculo-nerveux : intérêt de l’IRM

Arous

Riahi

Walha

et al. 2015

Pratique Neurologique - FMC

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Section: Discussionunclassified

Paralysie brutale du nerf oculomoteur commun révélant un conflit vasculo-nerveux : intérêt de l’IRM

Arous

Riahi

Walha

et al. 2015

Pratique Neurologique - FMC

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“…It seems more likely that the longlasting sixth nerve palsies described here were microvascular in origin rather than from inflammatory demyelination. Although the third nerve palsies described in ophthalmoplegic migraine may be different, it is of interest that the swelling and enhancement described in oculomotor migraine were intracisternal, whereas inflammatory third nerve lesions in general affect both intracisternal and more anterior regions of the third nerve (17).…”

Section: Discussionmentioning

confidence: 99%

The Aetiology and Management of Long-Lasting Sixth Nerve Palsy in Ophthalmoplegic Migraine

Manzouri¹,

Sainani²,

Plant

et al. 2007

Cephalalgia

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The features and management of two adult patients with ophthalmoplegic migraine and longlasting sixth nerve palsies are described. Both had had previous shorter episodes of diplopia following migraine-like headaches. One recovered following an injection of botulinum toxin to the medial rectus of her affected eye 11 months after the onset of diplopia. The other patient had previously had surgery for a consecutive divergent squint and required further squint surgery to realign his eyes 1 year after the onset of his sixth nerve palsy. Both botulinum toxin and squint surgery may be useful in the management of longstanding sixth nerve palsy in patients with ophthalmoplegic migraine. The aetiology of ophthalmoplegic migraine is discussed.

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“…Magnetic resonance angiography (MRA) and computer‐assistant tomography angiography (CTA) are relatively non‐invasive procedures that are becoming more mainstream due to the inherit risks associated with the use of cerebral arteriography. Although MRA has detected aneurysms as small as two to three millimetres, 25 the MRA is not as sensitive as cerebral arteriography in detecting aneurysms, particularly those less than five millimetres, which have been reported to be missed in about half the cases where a MRA alone was used 23 . In most cases, aneurysms causing third nerve palsy are five millimetres or greater, where the sensitivity of the MRA is significantly better with up to 97 per cent detection 23 …”

Section: Discussionmentioning

confidence: 99%

Pupil sparing incomplete third nerve palsy secondary to a cavernous sinus meningioma: challenges in management

Shechtman

Woods

Tyler

2007

Clinical and Experimental Optometry

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Background:The diagnosis of incomplete third nerve palsy can be clinically challenging because the aetiologies, as well as presentations, can be variable and subtle. The optometric clinician should be familiar with the association of third nerve palsy with compressive lesions, including the clinical presentations and management of these patients. Case report: We present a 68-year-old hypertensive male complaining of intermittent diplopia for the previous six months. Neurological examination revealed a mild deficit in adduction, reduced range of vertical motility and questionable ptosis, all limited to the OD. No pupillary involvement was noted. MRI revealed a cavernous sinus meningioma. The patient elected against surgery, choosing MRI every six months. Conclusion: Due to the subtle clinical presentation of incomplete pupil sparing third nerve palsy, this ophthalmoplegia may be easily overlooked. The associated aetiologies of palsy of cranial nerve three (CN3) carry increased risk of morbidity and mortality. Management should include evaluation for mass lesions and/or vascular aetiologies, regardless of pupillary involvement.Pupil sparing incomplete third nerve palsy secondary to a cavernous sinus meningioma: challenges in management

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Imaging evaluation of cranial nerves 3, 4, and 6

Cited by 15 publications

References 46 publications

Paralysie brutale du nerf oculomoteur commun révélant un conflit vasculo-nerveux : intérêt de l’IRM

Paralysie brutale du nerf oculomoteur commun révélant un conflit vasculo-nerveux : intérêt de l’IRM

The Aetiology and Management of Long-Lasting Sixth Nerve Palsy in Ophthalmoplegic Migraine

Pupil sparing incomplete third nerve palsy secondary to a cavernous sinus meningioma: challenges in management

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