Imaging Diagnosis—rathke's Cleft Cyst

Hasegawa, Daisuke; Uchida, Kazuyuki; Kobayashi, Masanori; Kuwabara, Takayuki; T, Ide; Ogawa, Fukie; Fujita, Michio; Orima, Hiromitsu

doi:10.1111/j.1740-8261.2009.01537.x

Cited by 14 publications

(20 citation statements)

References 14 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Choroid plexus cyst, 72,73 ependymal cyst, 74 and adenohypophyseal (Rathke's cleft) cyst 75,76 occur in dogs. These lesions appear similar in MR images, being thin‐walled cavitary lesions containing fluid that is hypointense in T1W images and hyperintense in T2W images, but their anatomic location will vary.…”

Section: Other Intracranial Cystsmentioning

confidence: 99%

“…These lesions appear similar in MR images, being thin‐walled cavitary lesions containing fluid that is hypointense in T1W images and hyperintense in T2W images, but their anatomic location will vary. Choroid plexus cysts were reported in the fourth ventricle in two dogs, 72,73 whereas adenohypophyseal cysts occupy the pituitary fossa, as they arise from the normal cleft between the neuro‐ and adenohypophysis 75,76 . Also, the central portion of an adenohypophyseal cyst does not null on FLAIR images because of its mucinous content 76 .…”

Section: Other Intracranial Cystsmentioning

confidence: 99%

“…Choroid plexus cysts were reported in the fourth ventricle in two dogs, 72,73 whereas adenohypophyseal cysts occupy the pituitary fossa, as they arise from the normal cleft between the neuro‐ and adenohypophysis 75,76 . Also, the central portion of an adenohypophyseal cyst does not null on FLAIR images because of its mucinous content 76 . The clinical signs associated with most intracranial cystic lesions are the result of impingement on adjacent neural structures.…”

Section: Other Intracranial Cystsmentioning

confidence: 99%

See 2 more Smart Citations

Magnetic Resonance Imaging of Intracranial Malformations in Dogs and Cats

Mackillop¹

2011

Vet Radiology Ultrasound

View full text Add to dashboard Cite

Intracranial malformations may occur because of an inherent developmental defect or secondary to in utero injury to the brain with subsequent hypoplasia and atrophy. They can cause neurologic deficits in growing animals, although some anomalies may not produce clinical signs until adulthood. Malformations of the brain include hydrocephalus, hydranencephaly/porencephaly, holoprosencephaly, corpus callosum agenesis/dysgenesis, lissencephaly, polymicrogyria, meningoencephalocele, intracranial cysts, cerebellar malformations, and hamartomas. These conditions are defined and reviewed with an emphasis on their features in magnetic resonance images.

show abstract

Section: Other Intracranial Cystsmentioning

confidence: 99%

Section: Other Intracranial Cystsmentioning

confidence: 99%

Section: Other Intracranial Cystsmentioning

confidence: 99%

See 1 more Smart Citation

Magnetic Resonance Imaging of Intracranial Malformations in Dogs and Cats

Mackillop¹

2011

Vet Radiology Ultrasound

View full text Add to dashboard Cite

show abstract

“…17 Cysts in the sellar region of human beings include Rathke cleft cysts, arachnoid cysts, dermoid and epidermoid cysts, 7 or oral radicular cysts. Cysts with a sellar location reported in dogs include Rathke cleft cysts, 8 cysts derived from the craniopharyngeal duct, 4 and cysts that result from the failed formation of the adenohypophysis, resulting in pituitary dwarfism. 4,11 Intracranial arachnoid cysts have also been reported in dogs, but these cysts were located in the quadrigeminal cistern and the cerebromedullary angle rather than the suprasellar space.…”

mentioning

confidence: 99%

“…Dogs with sellar and suprasellar masses can exhibit behavioral changes, 18 neurologic deficits, and endocrine dysfunction. Behavioral changes might include depression, 22,24 aggression, 8 lethargy, 6 anorexia, 24 polyphagia, 24 and polydipsia. 6 Neurologic deficits might include proprioceptive deficits, 6 circling, 6,24 and cranial nerve deficits, particularly related to compression of the optic chiasm.…”

mentioning

confidence: 99%

Sellar Xanthogranuloma in a Dog

et al. 2011

View full text Add to dashboard Cite

Abstract. A 7-year-old, intact male standard Poodle dog with hypothyroidism and atypical hyperadrenocorticism developed acute signs of lethargy, weakness, inappetence, vomiting, and diarrhea. Clinical signs progressed to hind limb proprioceptive deficits, aggressive behavior with obtundation, and an equivocal seizure. Necropsy revealed a mass in the sellar region that histologically consisted of multinucleated giant cells, macrophages, and hemosiderin-laden macrophages with fewer lymphocytes and plasma cells admixed with large regions of cholesterol cleft deposition, fibrin, and prominent Rosenthal fibers. Pituitary tissue was not identified on gross or histologic examination, but the mass was partially bordered by epithelial cells. The histologic characteristics are similar to changes described in the human medical literature as xanthogranuloma of the sellar region and xanthogranulomatous hypophysitis.Key words: Atypical hyperadrenocorticism; canine; craniopharyngioma; Rathke cleft cyst; xanthogranuloma of the sellar region; xanthogranulomatous hypophysitis. and polydipsia. 6 Neurologic deficits might include proprioceptive deficits, 6 circling, 6,24 and cranial nerve deficits, particularly related to compression of the optic chiasm. 16,18,22,24 Last, endocrine disease might manifest as hypothyroidism or diabetes insipidus. 16A 7-year-old, intact male standard Poodle dog presented to the Oklahoma State University Boren Veterinary Medical Teaching Hospital (Stillwater, Oklahoma) with a 1-year history of polyuria and polydipsia and a 1-week history of inappetence. The week before, the dog had been diagnosed with hypothyroidism on the basis of decreased total thyroxine (T4), and treatment with levothyroxine (0.01 mg/kg every 12 hr) had been instituted.Other than mild lumbosacral pain, physical examination findings were within normal limits. Because inappetence had coincided with initiation of thyroid supplementation, and euthyroid-sick syndrome was a differential diagnosis, treatment was discontinued to allow for future reassess- At the 1-month recheck appointment, the dog exhibited hair loss and lethargy consistent with previously diagnosed hypothyroidism. Repeat thyroid testing revealed a low free T4 (4.8, ref. interval: 8-40 pmol/l) with normal thyroidstimulating hormone and T4. Hypercholesterolemia had increased (435 mg/dl). Thyroid supplementation was reinstituted (levothyroxine, 0.02 mg/kg every 12 hr).Concurrently, to address the abnormal ACTH stimulation results seen on initial presentation, low-dose dexamethasone suppression testing was performed. Results were within normal limits, making typical hyperadrenocorticism less likely. Because of the persistent polyuria and polydipsia, additional tests were performed to assess other adrenocortical hormone levels. After exogenous ACTH stimulation, there were elevations in cortisol (191.8, ref. interval: 70.6-161.2 ng/ml), progesterone (6.57, ref. interval: 0.22-1.45 ng/ml), 17-OH progesterone (5.31, ref.interval: 0.25-2.63 ng/ml), and aldosterone (458.7, ref. ...

show abstract

Pituitary Gland

and¹,

Schwarz²

2011

Veterinary Computed Tomography

View full text Add to dashboard Cite

Imaging Diagnosis—rathke's Cleft Cyst

Cited by 14 publications

References 14 publications

Magnetic Resonance Imaging of Intracranial Malformations in Dogs and Cats

Magnetic Resonance Imaging of Intracranial Malformations in Dogs and Cats

Sellar Xanthogranuloma in a Dog

Pituitary Gland

Contact Info

Product

Resources

About