Idiotypic network, pathogenic autoantibodies and autoimmunity

Shoenfeld, Yehuda

doi:10.1111/j.1365-2249.1995.tb06159.x

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Active Proteinase-3 Vasculitis In Mice1

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“…Additionally, a string of immune network regulation was observed by the sequential appearance of Ab2 (mouse anti-human ANCA antibodies), then Ab3 (mouse anti-mouse anti-human ANCA antibodies), and finally Ab4 (mouse anti-mouse anti-mouse anti-human ANCA antibodies). This phenomenon has been explained previously [35]. The appearance of Ab3, a mouse anti-mouse antihuman ANCA antibody, having itself the specificity of the original human ANCA, is suggested as the reason for the perpetuation of the immune attack beyond the half life of the original passive transfer of human ANCA antibodies.…”

Section: Active Proteinase-3 Vasculitis In Micementioning

confidence: 69%