Identifying Patients Likely to Have Atopic Dermatitis

Farage, Miranda A.; Bowtell, Philip; Κατσαρού, Αλεξάνδρα

doi:10.2165/11530100-000000000-00000

Cited by 6 publications

(4 citation statements)

References 5 publications

(4 reference statements)

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“…Of potential utility for large‐scale screening in industry, postmarket surveillance, and epidemiological testing, a rapid algorithm containing only three questions was developed. Using the algorithm method, it was capable of identifying 88% of atopic individuals out of a population of sensitive‐skin patients (100).…”

Section: Sensitive Skin: Zeroing In On Biological Originmentioning

confidence: 99%

Sensitive skin: closing in on a physiological cause

Farage

Maibach

2010

Contact Dermatitis

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The phenomenon of 'sensitive skin' is a relatively recent complaint in which certain individuals report more intense and frequent adverse sensory effects than the normal population upon use of cosmetic (personal-care) products. Originally defined as a minority complaint, sensitive skin is now claimed by a majority of women in industrialized countries and nearly half of men. Sensitive skin is self-diagnosed and typically unaccompanied by any obvious physical signs of irritation, and the number of individuals who claim sensitivity has risen steadily with the number of consumer products targeted towards this supposedly uncommon group. Believed by many dermatologists, therefore, to be a 'princess and the pea' phenomenon, the problem of sensitive skin has largely avoided focussed research. Over the last few years, however, the evidence of documentable biophysical changes associated with the largely sensory symptoms of this disorder has accumulated, including some gained by improved methods of identifying subclinical signs of skin irritation. Although the understanding of the aetiology of this phenomenon is as yet incomplete, existing research now supports a biophysical origin for this disorder. Effective methods of diagnosis, intrinsic and extrinsic contributors to exaggerated neural sensitivity, and the specific mechanisms of the discomfort associated with the compliant are required, as are appropriate means of prevention and treatment.

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Section: Sensitive Skin: Zeroing In On Biological Originmentioning

confidence: 99%

Sensitive skin: closing in on a physiological cause

Farage

Maibach

2010

Contact Dermatitis

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“…Testing of the algorithm in a capable of identifying 88% of atopic individuals out of a population of sensitive-skin patients (101).…”

Section: Links Between Sensitive Skin and Immunologymentioning

confidence: 99%

Sensitive skin: new findings yield new insights

Farage¹,

Maibach²

2010

Textbook of Cosmetic Dermatology

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“…All patients in the atopic dermatitis group described themselves as having sensitive skin to at least some degree, with 80% claiming either moderately or very sensitive skin. By contrast, 64% of individuals in the control group described their skin as sensitive to some degree, with only 16% claiming either very or moderately sensitive skin [81]. Patients with atopic dermatitis were also significantly more likely to indicate a family history of sensitive skin than were non-sensitive individuals (68% to 24%, P = 0.004); 76% of atopic patients who claimed a family history identified a parent as having sensitive skin [81].…”

Section: Skin Sensitivitymentioning

confidence: 90%

A Body Divided: Toward Reunification of the Paradigm

Farage

Miller²,

Tzeghai³

et al. 2014

BJMMR

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Symptoms without obvious physical cause are commonly reported in medical practice; when chronic, they can have a significant influence on patients' well-being. When traditional medicine is unable to provide relief, sufferers of such conditions often turn to alternative therapies. Western medicine has historically viewed the body through a silo model, i.e, a whole consisting of disparate body systems with well-defined boundaries and little relevant interaction. This model ignores the myriad of interactive functions that each system must require and hinders understanding of syndromes for which etiology is not confined to one organ system, particularly those with a strong psychosocial component. In addition, this model is increasingly shown to be antiquated: recent evidence of Pavlovian conditioning of physiological processes (i.e., placebo and nocebo affects, immune system conditioning), physiological distinctions between multiple personalities, and the pervasive effects of psychosocial stress on every body system (down to the level of the genome) demand a new paradigm. As our appreciation expands of the innumerable interactions between body systems as well as those between all body systems and the mind, the human body is revealed to be a complex web of neurological, immunologic and endocrine interactions that in turn modulate a fluid epigenetic base. Firmly planted in the rationalistic viewpoint that is the foundation of Western medicine, but inclusive of the more wholistic (mind and body) view of Eastern medicine, a nexus model which views the body as the series of multi-connected, interacting physiological webs is essential to continued progress in medicine.

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Identifying Patients Likely to Have Atopic Dermatitis

Cited by 6 publications

References 5 publications

Sensitive skin: closing in on a physiological cause

Sensitive skin: closing in on a physiological cause

Sensitive skin: new findings yield new insights

A Body Divided: Toward Reunification of the Paradigm

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