“…Some authors have tried to explain the phenomenology of NDE by diverse physiological explanations such as anoxic brain damage (Rodin, 1980; Blackmore, 1993; Greyson, 1998; Els et al, 2004; Woerlee, 2005; Ammermann et al, 2007), hypoxia (Lempert et al, 1994), hypercapnia (Klemenc-Ketis et al, 2010), abnormal temporal lobe dysfunctions (Blanke et al, 2002, 2004; Britton and Bootzin, 2004; Blanke and Mohr, 2005; Arzy et al, 2006; Hoepner et al, 2013), administration of sedatives (Cobcroft and Forsdick, 1993; Osterman et al, 2001; Lopez et al, 2006), or sleep abnormalities (Nelson et al, 2006, 2007). However, to the best of our knowledge, previous studies on NDE after coma have not aimed at identifying differences in NDE characteristics depending on the etiology (i.e., traumatic, non-traumatic anoxic or non-traumatic other acute brain insults) of the prolonged loss of consciousness.…”