“…Considering the latest information about demyelinating defects and some remyelinating potentials illustrated here one still remain to know whether with delayed time after ODS damages, a sort of modus vivendi between astrocytes, oligodendrocytes, and neurons signals in making remyelination to return ODS thalamus or other CNS-damaged regions completely able to retrieve standard structures and functions. Possibly, similar repairs occurred in human ODS where most patients appeared to have a favorable clinical prognosis in the short-term post-hospitalization even though, retrospectively, there were and are still concerns for a range of 10 to 25% fatal outcomes [22,28,45,[107][108][109][110][111][112][113][114][115][116][117][118][119][120]123,259,260], or other neurologic defects [112,261], especially due other underlying disease states [45,[259][260][261][262].…”