Hypertrophic olivary degeneration after pontine hemorrhage

Asal, Neşe; Yılmaz, Ömer; Turan, Aynur; Yiğit, Hasan; Duymuş, Mahmut; Tekin, Evrim Karadağ

doi:10.1007/s00234-011-0897-4

Cited by 18 publications

(25 citation statements)

References 10 publications

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“…In HOD, diagnosis is usually made by MRI, and there is unilateral involvement in most patients; however, it can be bilateral in some patients (4)(5)(6)(7)(8)(9). In particular, ischemia, demyelization, and neoplasm should be considered in differential diagnosis (4)(5)(6)(7)(8)(9).…”

Section: Discussionmentioning

confidence: 99%

“…In particular, ischemia, demyelization, and neoplasm should be considered in differential diagnosis (4)(5)(6)(7)(8)(9). In HOD, increased signal intensity with or without olivary proliferation is observed at the ventral medulla on T2-weighted images (4-9).…”

Section: Discussionmentioning

confidence: 99%

“…Hypertrophic olivary degeneration (HOD) is a rarely encountered condition (4). It is often related to either dentate nucleus or contralateral cerebral pedicle and ipsilateral central tegmental tract lesions (5)(6)(7)(8)(9).…”

Section: Introductionmentioning

confidence: 99%

See 2 more Smart Citations

Traumatic Basilar Artery Dissection and Hypertrophic Olivary Degeneration

Canpolat¹,

Kumandaş²,

Esen³

et al. 2015

Erciyes Med J

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Head trauma is an important health issue during childhood and is the most frequent cause of mortality and morbidity during this period. Ischemic cerebral infarct following minor head traumas is seen at regions supplied by the middle cerebral artery, while the vertebra-basilar system is involved to a lesser degree. In this manuscript, we presented the case of a 7-month-old boy who presented with hemiparesis following head trauma, in which basilar artery dissection-related infarction and hypertrophic olivary degeneration were detected.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

See 1 more Smart Citation

Traumatic Basilar Artery Dissection and Hypertrophic Olivary Degeneration

Canpolat¹,

Kumandaş²,

Esen³

et al. 2015

Erciyes Med J

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“…Hypertrophic olivary degeneration is a rare condition, caused by the interruption of the neuronal connections of the dentatorubral olivary pathway, often referred to as the ‘Guillain Mollaret triangle' [6]. …”

Section: Discussionmentioning

confidence: 99%

“…Hypertrophic olivary degeneration is associated with late-onset neurological worsening after brainstem injury, but this has seldom been reported after stroke [6,7,8,9]. …”

Section: Introductionmentioning

confidence: 99%

Hypertrophic Olivary Degeneration after Pontine Hemorrhage: A Cause of Delayed Neurological Deterioration

Cordeiro

Tavares²,

Reimão³

et al. 2013

Cerebrovasc Dis

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Hypertrophic Olivary Degeneration Secondary to Neuro-Behçet’s Disease

et al. 2015

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A 22-year-old man presented with an acute altered mental status, left third cranial nerve palsy, ataxia, and right side weakness, all of which suggestive of brainstem encephalitis. Six months earlier, he had persistent fever, myalgia, and weight loss. Soon after fever resolution, oral and scrotal ulcers, usually painless, appeared accompanied by acneiform skin lesions in the chest and arms. At the time, antibiotic treatment led to partial regression of the lesions.Brain magnetic resonance imaging (MRI) showed hyperintense lesions on left midbrain, thalamus, and subcortical regions (Fig. 1). Cerebrospinal fluid analysis showed only mild lymphocytic pleocytosis. Pulse therapy with methylprednisolone 1 g/day for 7 days followed outstanding clinical improvement, although residual right hemiparesis and kinetic tremor remained. At discharge, he was on prednisone 20 mg/day and azathioprine 150 mg/day. Four months after therapy onset, a control brain MRI demonstrated resolution of most lesions along with signal alteration and enlargement of the olivary nuclei in the left medulla (Fig. 2a). At the time, he presented only minor oculopalatal myoclonus. Clinical stability and imaging pattern of the lesion suggested a benign finding, and no further investigation was pursued.Six years later, brain MRI showed an atrophic left midbrain peduncle with less evident olivary degeneration. In addition, there was significant atrophy of right cerebellar hemisphere (Fig. 2b) and shrinkage of midbrain (Fig. 2c, d). At that point, the patient had only mild right hemiparesis and oculopalatal myoclonus. Azathioprine 150 mg/day was his only medication, and he remains stable until this moment.

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Hypertrophic olivary degeneration after pontine hemorrhage

Cited by 18 publications

References 10 publications

Traumatic Basilar Artery Dissection and Hypertrophic Olivary Degeneration

Traumatic Basilar Artery Dissection and Hypertrophic Olivary Degeneration

Hypertrophic Olivary Degeneration after Pontine Hemorrhage: A Cause of Delayed Neurological Deterioration

Hypertrophic Olivary Degeneration Secondary to Neuro-Behçet’s Disease

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