Hypereosinophilic syndrome

Resnick, Myron E.; Myerson, Ralph M.

doi:10.1016/0002-9343(71)90262-2

Cited by 20 publications

(2 citation statements)

References 17 publications

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“…The title of hypereosinophilic syndrome has been coined (Hardy and Anderson, 1968;Resnick and Myerson, 1971) to describe a wide spectrum of disorders ranging from acute to chronic, benign to malignant, and involving from one to many organs. Hence, at one extreme is the benign disorder of transient pulmonary infiltration associated with eosinophilia (Loffler's syndrome) and at the other, a disseminated vasculitis.…”

Section: Discussionmentioning

confidence: 99%

Diffuse vasculitis, eosinophilia, and elevated antibody titre to measles virus

Bucknall

Doshi

1976

Postgraduate Medical Journal

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In the original description of serum sickness by Von Pirquet and Schick (1905) vasculitis was described as a feature. This was subsequently shown to be due to circulating immune complexes, which are known to be eosinotactic. The following report describes a case of serum sickness, occurring in a 14-year-old boy, characterized by diffuse vasculitis with communicating hydrocephalus and accompanied by a pronounced eosinophilia. A significantly raised antibody (IgG) titre to measles virus was demonstrated in his serum and indicated that this agent probably provided the initial antigenic stimulus.

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Section: Discussionmentioning

confidence: 99%

Diffuse vasculitis, eosinophilia, and elevated antibody titre to measles virus

Bucknall

Doshi

1976

Postgraduate Medical Journal

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“…An ascitis and hepatomegaly [10, 11] (on clinical exam and also shown by the abdominal echography of 01-27-2003), associated with edema and decreased prothrombin time (PT) 38% (range 70%–100%). These symptoms were ruled to be due to liver cirrhosis; therefore, the cytosine arabinoside was stopped because of its potential of liver toxicity and toxic encephalopathy.…”

Section: Case Reportmentioning

confidence: 99%

Clonal Hypereosinophilic Syndrome: Two Cases Report in Black Men from Sub-Saharan Africa and Literature Reviews

et al. 2011

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The first case is about a man of 60 years old suffering of hypereosinophilic syndrome (HES) developed since 1998. He presented chronic cough, insomnia, and negative parasitical test. We observed hypereosinophilia and fibroblastic hyperplasia at the bone marrow biopsy. Initially, hydroxyurea and α-interferon treatment failed. We proposed to him imatinib mesylate in May 2003. The FIP1L1-PDGFRA gene was detected. The second case is about a man of 34 years old seen in March 2002. First investigation concluded to CML. Progressively, eosinophil cells increased, and complications occurred as oedema syndrome, dyspnoea, and parietal chronic endocarditic fibrosis associated with pericarditis. In addition, a bowel obstruction happened and was cured by surgery. Bcr-abl fusion was negative, and FIP1L1-PDGFRA gene was detected after and imatinib mesylate was given. Actually, endocarditic fibrosis decreased. The two patients are in haematological and cytogenetic remission. We concluded that clonal HES is present in Africa, and imatinib mesylate is effective.

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Eosinophilic Endomyocarditis Complicating Acute Myocardial Infarction

Lie¹

1974

Arch Intern Med

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Hypereosinophilic syndrome

Cited by 20 publications

References 17 publications

Diffuse vasculitis, eosinophilia, and elevated antibody titre to measles virus

Diffuse vasculitis, eosinophilia, and elevated antibody titre to measles virus

Clonal Hypereosinophilic Syndrome: Two Cases Report in Black Men from Sub-Saharan Africa and Literature Reviews

Eosinophilic Endomyocarditis Complicating Acute Myocardial Infarction

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