Hypercalcaemia in a dog with primary hypothyroidism : clinical communication

Lobetti, Remo

doi:10.4102/jsava.v82i4.81

Cited by 6 publications

(1 citation statement)

References 8 publications

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“…Causes such as hypoadrenocorticism and vitamin D toxicity were excluded based on ACTH stimulation and history 40 . Interestingly, hypercalcaemia has been reported in association with both congenital 22 and acquired 41 canine hypothyroidism. However, this was deemed unlikely given hypercalcaemia developed after initiation of levothyroxine treatment when the dog was euthyroid.…”

Section: Discussionmentioning

confidence: 99%

Juvenile primary acquired hypothyroidism in a dog with suspected renal dysplasia

Parton,

Atkins,

Boag

et al. 2023

Vet Record Case Reports

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A 1‐year 3‐month‐old, female, neutered crossbreed dog presented for investigation of polyuria, polydipsia and azotaemia. Subsequent biochemistry, endocrine testing and abdominal ultrasound identified hypercholesterolaemia, thyroglobulin autoantibody‐positive hypothyroidism, and IRIS stage II non‐hypertensive, non‐proteinuric suspected renal dysplasia, with a concurrent Escherichia coli urinary tract infection. The patient responded well clinically to levothyroxine treatment, with improvement in hypercholesterolaemia, azotaemia and demeanour at home. At follow‐up biochemistry assessment, hyperphosphataemia and hypercalcaemia (total and ionised) were documented, presumed to be driven by renal insufficiency and renal secondary hyperparathyroidism, although this aetiology was not definitively proven. Normalisation of these values was noted following renal diet implementation and addition of phosphate binders. This case highlights acquired primary hypothyroidism with evidence of thyroiditis in a juvenile dog with suspected congenital renal dysplasia.

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Section: Discussionmentioning

confidence: 99%