HPV-associated diseases

Ljubojević, Suzana; Skerlev, Mihael

doi:10.1016/j.clindermatol.2013.08.007

Cited by 112 publications

(84 citation statements)

References 70 publications

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“…The involvement of cutaneous HPV genotypes in nonmelanoma skin cancer (NMSC) is still not established and the development of NMSC seems to be the result of multiple events, being UV irradiation the key factor, and HPV infection a co-factor (Ljubojevic and Skerlev 2014). The role of cutaneous HPV E6 and E7 proteins in tumorigenesis has been poorly clarified.…”

Section: Discussionmentioning

confidence: 99%

Human papillomavirus E6 and E7 oncoproteins affect the expression of cancer-related microRNAs: additional evidence in HPV-induced tumorigenesis

Chiantore

Mangino

Iuliano

et al. 2016

J Cancer Res Clin Oncol

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Section: Discussionmentioning

confidence: 99%

Human papillomavirus E6 and E7 oncoproteins affect the expression of cancer-related microRNAs: additional evidence in HPV-induced tumorigenesis

Chiantore

Mangino

Iuliano

et al. 2016

J Cancer Res Clin Oncol

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“…17 Clinically, they most commonly involve exposed surfaces, usually on the dorsal aspect of the hands and fingers. Uncommonly, lesions may appear in covered areas, such as axillae, groins, or genital areas.…”

Section: Verruca Vulgarismentioning

confidence: 99%

Viral infections of the folds (intertriginous areas)

Adışen

Önder

2015

Clinics in Dermatology

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“…The latter group comprising HPVs16 /18/ 31/ 33/ 45/ 51/ 52/ 58 etc. is associated with more than 90% of the mucosal cancers including cervical, anal, oropharyngeal, penile etc [12]. Several studies have been conducted in the past and many more are ongoing to explore this virus mediated oncogenic transformation and mechanism [13][14][15][16].…”

Section: Hpv and Diseasesmentioning

confidence: 99%

Human Papilloma Virus (HPV) Vaccines: Scope and Implications

Singh¹,

Satapathy²,

Singh³

2016

Transl Med (sunnyvale)

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High risk human papilloma virus has often been associated with cancer onset and progression by the virtue of the viral oncogenes. Genital cancers and Oropharyngeal cancers are the two leading carcinomas often induced by HPV infection. These viral encoded oncogenes (e.g. E6/E7/E8) are known for their ability to integrate, recombine and interfere with the host cellular genes. Although significant work has been undertaken and several more in progress in understanding the disease biology, yet attaining an effective and long term protection from HPV associated cancers remains unachieved. Lack of any drug based therapy or post-infection treatment strategy along with limited commercially available vaccines (e.g. Gardasil and Cervarix) have crippled our efforts to curb the disease manifestation cancers, remains. Therefore, in this review we aim to address the contemporary rational of HPV vaccine designing and development. We have also analyzed the scope and future perspectives of the commercially available vaccine regimes.

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HPV-associated diseases

Cited by 112 publications

References 70 publications

Human papillomavirus E6 and E7 oncoproteins affect the expression of cancer-related microRNAs: additional evidence in HPV-induced tumorigenesis

Human papillomavirus E6 and E7 oncoproteins affect the expression of cancer-related microRNAs: additional evidence in HPV-induced tumorigenesis

Viral infections of the folds (intertriginous areas)

Human Papilloma Virus (HPV) Vaccines: Scope and Implications

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