HIV-positiver Patient mit multiplen Ulzerationen

Körber, Andreas; Dissemond, Joachim; Hillen, Uwe; Goos, M.; Esser, S.

doi:10.1007/s00105-003-0619-x

Cited by 5 publications

(3 citation statements)

References 11 publications

(10 reference statements)

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“…Firstly, HIV is thought to be the most variable virus, and its clinical characteristics alone are not sufficient to explain the outcomes of infection. In addition, it has subtypes with distinct geographic distribution and recombination can occur between and within subtypes (Korber, 2003). Thus creating a further increase in the genetic diversity in many parts of the world and thereby a complex challenge to control.…”

Section: Circulating Recombinants Forms Of Hiv In Malimentioning

confidence: 99%

HIV Prevention Needs Epidemiological Data

Tounkara¹,

Koné²,

Diarra³

et al. 2011

HIV-infection - Impact, Awareness and Social Implications of Living With HIV/AIDS

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Section: Circulating Recombinants Forms Of Hiv In Malimentioning

confidence: 99%

HIV Prevention Needs Epidemiological Data

Tounkara¹,

Koné²,

Diarra³

et al. 2011

HIV-infection - Impact, Awareness and Social Implications of Living With HIV/AIDS

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“…Oft finden sich enoral Plaque muqueuses (Abbildung 2). In bis zu 7 % aller sekundären Syphilisfälle dieser Population zeigt sich das klinische Erscheinungsbild einer Lues maligna, die eine akzeleriert verlaufende granulomatöse und nekrotisierende Variante der Syphilis speziell bei HIV‐Koinfizierten darstellt (Abbildung 3) [13, 14, 15]. Eine exakte ätiologische Klärung ob eine Immundefizienz oder andere Faktoren für das Auftreten einer Lues maligna primär verantwortlich sind, konnte bislang nicht erfolgen.…”

Section: Frühsyphilisunclassified

Syphilis bei HIV‐Koinfektion

Körber

Dissemond

Lehnen

et al. 2004

J Deutsche Derma Gesell

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In recent years a rising incidence of syphilis has been observed, especially in the population of homosexual men. Because of altered sexual behavior in terms of increased promiscuity paralleled by decreased use of condoms and the fact that a syphilis infection increases the susceptibility to HIV coinfection, the incidence of HIV is also rising once again in this population. In patients with HIV coinfection, the course of syphilis is often atypical or dramatic. Stage-specific features suggesting coinfection include prolonged primary ulcers persisting well into the secondary stage, numerous atypical cutaneous findings in the second stage and a rapid progression from stage to stage. The diagnosis of syphilis may be more difficult because of false positive or false negative serological findings in patients with HIV coinfection. Whether or not the CNS is more often involved is this patient group has not been established by prospective studies and remains controversial. However, WHO and CDC recommendations include evaluation of the CSF in HIV-infected patients with either late syphilis or when the time course is unknown period. There is worldwide agreement on the therapy of syphilis in patients with HIV coinfection. Patients with early syphilis should be treated with 2.4 benzathine penicillin i.m. once or twice; patients with late syphilis, twice or three times. Patients presenting with clinical or serological signs of neurosyphilis require 18-24 million IU penicillin i.v. daily for at least 2 weeks.

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“…Bei gleichzeitig bestehender HIV-Koinfektion zeigt sich gehäuft die Lues maligna, die durch ulzerierende Syphilide der Haut gekennzeichnet ist(18). Alle Syphilide im Sekundärstadi-um sind potenziell infektiös.kurzgefasst: Die Syphilis verläuft in verschiedenen Stadien.…”

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