Histological Types of Gastric Cancer and its Relationship with Intestinal Metaplasia

Muñóz, Núbia; Matko, I

doi:10.1007/978-3-642-80686-5_9

Cited by 43 publications

(3 citation statements)

References 7 publications

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“…These clinically extremely important changes in serum PG levels are due to the above-mentioned unique distribution of PGproducing cells in gastric mucosal epithelia. In addition, the results of past pathological and epidemiological studies have shown that a strong correlation exists between chronic atrophic gastritis and differentiated gastric cancer development, and thus, chronic atrophic gastritis is considered to be a precancerous lesion (24)(25)(26)(27) …”

Section: Pepsinogen Testingmentioning

confidence: 99%

Serum Pepsinogen and Gastric Cancer Screening

et al. 2007

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Section: Pepsinogen Testingmentioning

confidence: 99%

Serum Pepsinogen and Gastric Cancer Screening

et al. 2007

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“…Generally, the ratio has been close to 1.00 in age groups under 40-50 years, but higher in the older age groups [4]. Generally, higher male-to-female ratios have been reported for the intestinal than diffuse histopathologies [4,9,15]. Gastric cancer is largely a disease of the older age groups in most countries [8].…”

Section: Age and Sex Distributionmentioning

confidence: 99%

The epidemiology of gastric cancer

2002

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“…Such markers include atrophic gastritis and intestinal metaplasia. [24][25][26] Others can be seen at a histochemical level. A significant risk predictor is the anomalous expression of blood group antigens in mucin, especially those of the Lewis group.…”

Section: Epidemiological Considerationsmentioning

confidence: 99%

Occupation and gastric cancer

Raj

Mayberry

Podas³

2003

Postgraduate Medical Journal

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Gastric cancer is a cause of significant morbidity and mortality. There are several risk factors, with occupation emerging as one of these. There is considerable evidence that occupations in coal and tin mining, metal processing, particularly steel and iron, and rubber manufacturing industries lead to an increased risk of gastric cancer. Other “dusty” occupations—for example, wood processing, or work in high temperature environments have also been implicated but the evidence is not strong. The mechanism of pathogenesis of gastric cancer is unclear and the identification of causative agents can be difficult. Dust is thought to be a contributor to the pathological process, but well known carcinogens such as N-nitroso compounds have been detected in some environments. Further research on responsible agents is necessary and screening for detection of precursor gastric cancer lesions at the workplace merits consideration.

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Histological Types of Gastric Cancer and its Relationship with Intestinal Metaplasia

Cited by 43 publications

References 7 publications

Serum Pepsinogen and Gastric Cancer Screening

Serum Pepsinogen and Gastric Cancer Screening

The epidemiology of gastric cancer

Occupation and gastric cancer

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