Hepatitis E as a cause of acute non-A, non-B hepatitis

Abstract

Hepatitis E seems to be responsible for most epidemics of non-A, non-B hepatitis in developing countries. While hepatitis E is not endemic in the United States, it should be considered in the differential diagnosis of the traveler with non-A, non-B hepatitis. We describe a patient with acute hepatitis E, confirmed by serologic testing, after recently returning from India and discuss current knowledge about the pathogenesis, clinical features, and diagnosis of hepatitis E.

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“…Alcohol increases HDL cholesterol so it is involved in tumors of high waste of cholesterol, and physical exercise increases the waste of cholesterol. As for acid diet and alkaline diet, the activation or deactivation of somatostatin in the digestive tracts is described [11][12][13], it increases itself the thyroid gland activity and the production of thyroid hormones, so a continuous acid or alkaline diet could influence the misbalance of the waste of cholesterol. On the other hand a regression of breast cancer has been described by Dr Di Bella G with somatostatin treatment.…”