Henoch-Schonlein purpura secondary to trauma.

Abstract

Computed tomography in childhood epilepsy Sir, Children attending the school at the David Lewis Centre for Epilepsy are a selected group, as with few exceptions they are residential pupils from various parts of the United Kingdom and therefore are likely to have severe and often multiple handicaps. It seemed probable that a group of such children might show a high incidence of significant abnormalities on computed tomography. The children attending the school at the David Lewis Centre and examined by computed tomography over several years numbered 222; there were 154 boys and 68 girls, aged 7 to 19 years. The results showed that 152 were normal. There were 28 with evidence of some degree of generalised atrophy and 30 with focal atrophy. Nine showed calcification and in three the findings were compatible with a cerebral tumour. Abnormalities in a third of the children is in agreement with the findings of Bachman et a1' and Yang et al.2 Bachman et al studied 98

Henoch-Schönlein purpura nephritis: an update

“…Indeed, several drugs such as cipro¯oxacin, acetylsalicylic acid, vancomycin, carbidopa/levodopa, cocaine, acetylcholinesterase inhibitors, carbamazepine and streptokinase [9,21,23,42,57,60,63,67,77] have been implicated in HSP induction. HSPN has also been occasionally reported in association with cancer [6], blunt trauma [80], monoclonal IgA gammopathy [22], Wiskott-Aldrich syndrome carriers [49], chronic alcoholic liver disease [28] and with alpha-1 antitrypsin de®ciency [25].…”