2004
DOI: 10.1111/j.1538-7836.2004.00956.x
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Hemostatic and hematological abnormalities in gain‐of‐function fps/fes transgenic mice are associated with the angiogenic phenotype

Abstract: To cite this article: Sangrar W, Senis Y, Samis JA, Gao Y, Richardson M, Lee DH, Greer PA. Hemostatic and hematological abnormalities in gain-of-function fps/fes transgenic mice are associated with the angiogenic phenotype.

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Cited by 3 publications
(1 citation statement)
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“…Furthermore, FES knockout (FES -/-) mice demonstrate dysregulation of signal transducer and activator of transcription 3 (STAT3) (13), which plays a vital role in the pathophysiology of sepsis by initiating endothelial dysfunction, vasoplegia, coagulopathy, and multi-organ failure (14,15). FES may also contribute to thrombin formation (16), which is known to contribute to hypercoagulation during the early inflammatory stage of sepsis (17).…”
Section: Introductionmentioning
confidence: 99%
“…Furthermore, FES knockout (FES -/-) mice demonstrate dysregulation of signal transducer and activator of transcription 3 (STAT3) (13), which plays a vital role in the pathophysiology of sepsis by initiating endothelial dysfunction, vasoplegia, coagulopathy, and multi-organ failure (14,15). FES may also contribute to thrombin formation (16), which is known to contribute to hypercoagulation during the early inflammatory stage of sepsis (17).…”
Section: Introductionmentioning
confidence: 99%