Hemorrhagic ocular complications associated with the use of systemic thrombolytic agents

Chorich, Louis J.; Derick, Robert J.; Chambers, Robert B.; Cahill, Kenneth V.; Quartetti, Eric J; Fry, John; Bush, Charles A.

doi:10.1016/s0161-6420(98)93023-8

Cited by 58 publications

(34 citation statements)

References 18 publications

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“…In addition to overtreatment with anticoagulants, some ocular issues such as age-related macular degeneration with neovascularization and nanophthalmos are risk factors for this uncommon complication. [94][95][96][97][98] Four patients with age-related macular degeneration who were on phenprocomoun or heparin developed unilateral ACAG after massive intraocular bleeding. Three of the four eyes were blind within a few months even with surgical evacuation of blood.…”

Section: Respiratory System Agentsmentioning

confidence: 99%

Non-steroidal drug-induced glaucoma

Razeghinejad

Pro

Katz

2011

Eye

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Numerous systemically used drugs are involved in drug-induced glaucoma. Most reported cases of non-steroidal drug-induced glaucoma are closed-angle glaucoma (CAG). Indeed, many routinely used drugs that have sympathomimetic or parasympatholytic properties can cause pupillary block CAG in individuals with narrow iridocorneal angle. The resulting acute glaucoma occurs much more commonly unilaterally and only rarely bilaterally. CAG secondary to sulfa drugs is a bilateral non-pupillary block type and is due to forward movement of iris-lens diaphragm, which occurs in individuals with narrow or open iridocorneal angle. A few agents, including antineoplastics, may induce open-angle glaucoma. In conclusion, the majority of cases with glaucoma secondary to non-steroidal medications are of the pupillary block closed-angle type and preventable if the at-risk patients are recognized and treated prophylactically.

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Section: Respiratory System Agentsmentioning

confidence: 99%

Non-steroidal drug-induced glaucoma

Razeghinejad

Pro

Katz

2011

Eye

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“…[1][2][3][4]7 Our patient, who presented in an advanced stage of visual loss, had a recurrent suprachoroidal bleed but was eventually free of pain and was therefore managed conservatively.…”

Section: Commentmentioning

confidence: 89%

Polypoidal choroidal vasculopathy causing massive suprachoroidal haemorrhage

Tan

Wong

Lim

et al. 2006

Eye

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“…1,2,9-12 Systemic causes usually result from bleeding disorders and include: haemophilia, von Willebrand's disease, heparin treatment, thrombolytic treatment and liver disease. [13][14][15][16] Other systemic causes described are anaemia, leukaemia, sickle cell disease, and hypertension. 2,3 Meningococcal infection is caused by the organism, Neisseria meningitidis, a gram negative diplococcus.…”

Section: Commentmentioning

confidence: 99%

Spontaneous orbital haemorrhage in purpura fulminans secondary to meningococcal septicaemia

Singh¹

2002

Eye

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Hemorrhagic ocular complications associated with the use of systemic thrombolytic agents

Cited by 58 publications

References 18 publications

Non-steroidal drug-induced glaucoma

Non-steroidal drug-induced glaucoma

Polypoidal choroidal vasculopathy causing massive suprachoroidal haemorrhage

Spontaneous orbital haemorrhage in purpura fulminans secondary to meningococcal septicaemia

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