Hemichorea (Hemiballismus) Without Lesions in the Corpus Luysii

Putaminal hemorrhage accompanied by hemichorea-hemiballism.

“…Reviewing human cases of hemichorea, Martin inferred that choreic movements resulted when the globus pallidus was released from the inhibitory influence of the subthalamic nucleus. 6 Results of an acute putaminal lesion suggest that the putamen similarly inhibits the globus pallidus and that removal of this inhibition leads to hemichorea, 7 as demonstrated in the present case.…”

Lacunes.

“…Strength, sensation, and reflexes were normal. Review of the patho-anatomical findings in other documented cases of hemichorea-hemiballismus reveals that many are consistent with lacunar infarcts in the subthalamic nucleus 20,7B striatum, 37,76 and thalamus, 19,76~77 although the vascular pathology has not been defined.…”

Hemiballism-Hemichorea and Posterior Communicating Artery Stenosis

“…These lesions can either be ischemic [4] or hemorrhagic [3,5], but underlying vascular pathology has been rarely investigated. Previous autopsy studies have described extensive atherosclerotic lesions in arteries including the posterior cerebral artery or the posterior communicating artery in some patients, but the brain lesions examined usually extended far beyond the subthalamic nucleus [6]. We herewith present a patient with subthalamic infarction producing HB-HC, which was related to focal stenosis in the posterior communicating artery.…”