Abstract:Background H. pylori is the most prevalent bacterial infection in the world, and its crucial virulence component CagA is the primary cause of gastric cancer. Mitophagy is a form of selective autophagy that eliminates damaged mitochondria and is essential for some viruses and bacteria to evade the immune system. However, the potential impact of H.pylori CagA in the crosstalk between mitophagy and NLRP3 inflammasome is not completely known.Objective In this study, we aimed to understand the impact of H. pylori … Show more
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