Heat Illness during Working and Preventive Considerations from Body Fluid Homeostasis

Kamijo, Yoshi‐ichiro; Nosé, Hiroshi

doi:10.2486/indhealth.44.345

Cited by 14 publications

(10 citation statements)

References 65 publications

(76 reference statements)

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“…Heat stress in commercial kitchens can be due to high air temperature, humidity, radiant heat and airflow [ 1 ]. Thermal strain induced by such occupations can lead to health risks like acute heat stroke, blood circulation problems, and discomfort at work place [ 2 ]. Heat stress is still the most neglected occupational hazard in tropical and subtropical countries [ 3 ].…”

Section: Introductionmentioning

confidence: 99%

Heat and PAHs Emissions in Indoor Kitchen Air and Its Impact on Kidney Dysfunctions among Kitchen Workers in Lucknow, North India

Singh¹,

Kamal²,

Mudiam³

et al. 2016

PLoS ONE

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Indoor air quality and heat exposure have become an important occupational health and safety concern in several workplaces including kitchens of hotels. This study investigated the heat, particulate matter (PM), total volatile organic compounds (TVOCs) and polycyclic aromatic hydrocarbons (PAHs) emissions in indoor air of commercial kitchen and its association with kidney dysfunctions among kitchen workers. A cross sectional study was conducted on 94 kitchen workers employed at commercial kitchen in Lucknow city, North India. A questionnaire-based survey was conducted to collect the personal and occupational history of the kitchen workers. The urine analysis for specific gravity and microalbuminuria was conducted among the study subjects. Indoor air temperature, humidity, wet/ dry bulb temperature and humidex heat stress was monitored during cooking activities at the kitchen. Particulate matter (PM) for 1 and 2.5 microns were monitored in kitchen during working hours using Hazdust. PAHS in indoor air was analysed using UHPLC. Urinary hydroxy-PAHs in kitchen workers were measured using GC/MS-MS. Higher indoor air temperature, relative humidity, PM1 and PM2.5 (p<0.001) was observed in the kitchen due to cooking process. Indoor air PAHs identified are Napthalene, fluorine, acenaphthene, phenanthrene, pyrene, chrysene and indeno [1,2,3-cd) pyrene. Concentrations of all PAHs identified in kitchen were above the permissible OSHA norms for indoor air. Specific gravity of urine was significantly higher among the kitchen workers (p<0.001) as compared to the control group. Also, the prevalence of microalbuminuria was higher (p<0.001) among kitchen workers. Urinary PAH metabolites detected among kitchen workers were 1-NAP, 9-HF, 3-HF, 9-PHN and 1-OHP. Continuous heat exposure in kitchens due to cooking can alter kidney functions viz., high specific gravity of urine in kitchen workers. Exposure to PM, VOCs and PAHs in indoor air and presence of urinary PAHs metabolites may lead to inflammation, which can cause microalbuminuria in kitchen workers, as observed in the present study.

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Section: Introductionmentioning

confidence: 99%

Heat and PAHs Emissions in Indoor Kitchen Air and Its Impact on Kidney Dysfunctions among Kitchen Workers in Lucknow, North India

Singh¹,

Kamal²,

Mudiam³

et al. 2016

PLoS ONE

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“…Thus, the result is considered to be caused by physical work in the hot environment 31) . We observed high prevalence of individual protective measures against hot environment to work comfortably in summer 28,29) in the unexposed to cold group. The most individual measures were drinking water at short intervals (77.9%) and wearing the high hygroscopicity clothes (75.6%).…”

Section: Fig 6 Discomfort Index In the Workhops In Summermentioning

confidence: 78%

Subjective Symptoms of Female Workers Sorting Goods in Summer

2011

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Subjective musculoskeletal symptoms are more frequently complained about in cold store work and in related conditions than those experienced in normal temperature work. This cross sectional study was undertaken to evaluate the effects of indoor cooling and cold storage goods on the prevalence of subjective symptoms in summer. Female workers sorting cold storage goods (exposed group) were the main subjects of this study (n=47). We also included a group of female workers engaged sorting dry goods as the unexposed to cold group (n=86). Work load for the two groups were estimated according to the recommended criteria. A selfadministered questionnaire covering age, occupational career, smoking, alcohol drinking and physical exercise, present or past history of diseases, individual protective measures against cold or heat, and subjective symptoms (60 items) was used. The air temperature of the site at the start of working time for the workers sorting cold storage goods was 22.2°C which was significantly lower than those measured for the other two work places (25.4°C and 25.4°C) of the unexposed to cold group. Environmental temperatures at the foot level at the sorting workshop of cold storage goods and dry goods were ca.16°C and 26°C all day, respectively. The surface temperatures of cold storage goods were between -2.8°C and 9.4°C. The surface temperature of dry goods was 26.5°C. Among the working characteristic items, only daily working hours in the exposed group (5.6 ± 0.6 h) were significantly longer than those in the unexposed to cold group (4.6 ± 0.9 h) (p<0.01). The prevalence rates of finger cold sensation, stiffness in the fingers, pain in the wrist, pain in the elbow, back dullness, back pain, low-back cold sensation, foot cold sensation and pain in the foot in the exposed group were significantly higher than those in the unexposed to cold group (p<0.05 or p<0.01). Pain in the fingers, numbness in the fingers, pain in the foot and foot numbness due to the cold in the exposed group were significantly higher than those in the unexposed to cold group (p<0.05 or p<0.01). These results suggest that indoor cooling and/or job activities related to cold storage goods could, to some extent, affect peripheral circulatory disturbances; and it could be regarded as a factor related to musculoskeletal symptoms among the exposed workers.

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“…Roles of fluid intake for the recovery of body fluid after and during prolonged exercise under heat-stressed conditions have been well described [105][106][107][108][109] . Fluid intake is also required for reducing hemoconcentration during orthostatic stress.…”

Section: Rehydrationmentioning

confidence: 99%

Heat stress and orthostatic tolerance

Yamazaki

2012

JPFSM

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In healthy individuals, exercise and nonexercise (i.e. passive body heating) heat stress induces vulnerability in the cardiovascular system, and is apt to cause hypotension during upright posture, resulting in a reduction of orthostatic tolerance. Reduced orthostatic tolerance is linked to multiple physiological mechanisms including 1) a redistribution of blood flow from central parts of the body to skin, 2) an increase in leg venous compliance, 3) altered baroreflex function, 4) an attenuated venoarterial response in the lower extremities, and 5) a decrease in plasma volume. A combination of countermeasures such as cooling of the body, rehydration, and heat acclimation, can improve orthostatic tolerance in a hot environment. The purpose of this review was to summarize findings investigating the causes of and preventive approaches to heat stress-induced orthostatic intolerance.

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Heat Illness during Working and Preventive Considerations from Body Fluid Homeostasis

Cited by 14 publications

References 65 publications

Heat and PAHs Emissions in Indoor Kitchen Air and Its Impact on Kidney Dysfunctions among Kitchen Workers in Lucknow, North India

Heat and PAHs Emissions in Indoor Kitchen Air and Its Impact on Kidney Dysfunctions among Kitchen Workers in Lucknow, North India

Subjective Symptoms of Female Workers Sorting Goods in Summer

Heat stress and orthostatic tolerance

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