Head-shaking nystagmus in patients with unilateral peripheral vestibular lesions

Hain, Timothy C.; Fetter, M.; Zee, David S.

doi:10.1016/s0196-0709(87)80017-0

Cited by 308 publications

(119 citation statements)

References 15 publications

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“…With standard lighting, the clinical examination identifies overt deficits in smooth pursuit and saccadic eye movements (normal/abnormal), spontaneous and gaze holding nystagmus at 30 degrees from the midline (present/absent) (Hall and Herdman, 2006). The use of Frenzel Goggles enables observation of spontaneous nystagmus, gaze evoked nystagmus and head shaking nystagmus with vision removed, and if identified, is indicative of central dysfunction if suppression of nystagmus does not occur when a light is switched on within the Frenzel Goggles (Hain et al, 1987;Hall and Herdman, 2006). for impulses applied by an assessor in the horizontal plane at 40ms, 60ms and 80ms and identifies any asymmetry between left and right responses (Yang et al, 2016).…”

Section: Methodsmentioning

confidence: 99%

A prospective investigation of changes in the sensorimotor system following sports concussion. An exploratory study

Hides

Smith

Mendis

et al. 2017

Musculoskeletal Science and Practice

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Section: Methodsmentioning

confidence: 99%

A prospective investigation of changes in the sensorimotor system following sports concussion. An exploratory study

Hides

Smith

Mendis

et al. 2017

Musculoskeletal Science and Practice

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“…In peripheral vestibulopathy, HSN may occur from the velocity storage mechanism and Ewald's second law which states that excitatory vestibular inputs are more effective than inhibitory ones. 34,35 The nodulus and ventral uvula project to the vestibular nuclei and inhibit the velocity-storage mechanism of the VOR. 3,24 Because the effect of the nodulus on velocity-storage is unilateral, 24 unilateral or asymmetrical nodular lesion would generate asymmetry in the velocity storage, which would increase with horizontal head-shaking and induce the nystagmus or augment the spontaneous nystagmus.…”

Section: Soo Moon Et Al Isolated Nodular Infarction 489mentioning

confidence: 99%

Isolated Nodular Infarction

Moon

Kim

Choi

et al. 2009

Stroke

127

112

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Background and Purpose-Isolated nodular infarction has rarely been described in human. The purpose of this study is to report clinical and laboratory findings of isolated nodular infarction. Methods-Eight patients with isolated nodular infarction were recruited from 6 hospitals in Korea. All patients underwent a complete and standardized neurotological evaluation including ocular torsion, bithermal caloric tests, and rotatory chair test in addition to MRI and MR angiography. Results-All patients presented with isolated vertigo and moderate to severe imbalance. The most common manifestation was unilateral nystagmus and falling in the opposite direction, which mimicked peripheral vestibulopathy. Six patients had unilateral lesion, and 2 showed bilateral lesions. The direction of the spontaneous nystagmus was all ipsilesional in the unilateral lesion. However, head impulse and bithermal caloric tests were normal. Other findings include periodic alternating nystagmus, perverted head shaking nystagmus, paroxysmal positional nystagmus, and impaired tilt suppression of the postrotatory nystagmus. Hypoplasia of the ipsilesional vertebral artery was the only abnormal finding on MR angiography in 3 patients. The prognosis was excellent. Conclusions-Isolated

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“…A head impulse test was then performed and judged as positive to the left. A head shaking-induced nystagmus test using infrared goggles was performed next and judged as positive with approximately 10 beats of nystagmus to the right [17][18][19][20]. It was determined that the BPPV was resolved and a new physical therapy diagnosis of Left Unilateral Vestibular Hypofunction (L-UVH) was made.…”

Section: Session 3-(4 Days After Initial Consultation)mentioning

confidence: 99%

Differential diagnosis and clinical decision-making in physical therapy: Case report of a complex dizzy patient

Johnson¹,

Gowda²,

Alharbi³

2017

Phys Ther Rehabil

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Background: This case report describes the physical therapy differential diagnostic and clinical decisionmaking processes utilized in the management of a complex dizzy patient. Case presentation: A 55 year-old female patient with a congenital eye defect was initially diagnosed with left horizontal cupulolithiasis that was converted to horizontal canalithiasis using a modified liberatory maneuver. After resolving the left horizontal canalithiasis, the patient was diagnosed with left unilateral vestibular hypofunction, which was successfully treated by the physical therapist. Discussion: Over a period of 23 days and 5 physical therapy sessions, the patient management approach included examination (subjective and physical), evaluation of findings, physical therapy diagnosis, prognosis, plan of care, intervention, and evaluation of outcomes. Conclusion: This case report demonstrates the need for physical therapists to incorporate a comprehensive patient management approach in order to efficiently maximize patient outcomes.

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Head-shaking nystagmus in patients with unilateral peripheral vestibular lesions

Cited by 308 publications

References 15 publications

A prospective investigation of changes in the sensorimotor system following sports concussion. An exploratory study

A prospective investigation of changes in the sensorimotor system following sports concussion. An exploratory study

Isolated Nodular Infarction

Differential diagnosis and clinical decision-making in physical therapy: Case report of a complex dizzy patient

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