Hämatogene Kontaktekzeme durch Nahrungsmittel

Erdmann, Stephan; Werfel, Thomas

doi:10.1007/s00105-005-1079-2

Cited by 20 publications

(8 citation statements)

References 38 publications

(11 reference statements)

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“…Primarily hematogenously acting contact allergens include nickel sulfate, chromium compounds, parabens, balsam of Peru, and a variety of pharmaceutical agents .…”

Section: Differential Diagnoses Of Contact Dermatitismentioning

confidence: 99%

Contact dermatitis: relevant differential diagnoses, simulators, and variants

Kränke

Schuster²

2015

J Deutsche Derma Gesell

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SummarySimilar to all eczematous disorders, irritant and allergic contact dermatitis are primarily defined by their morphology. In addition, the diagnosis of this specific reaction pattern on the skin (and mucous membranes) also requires prior exposure (direct or indirect) to an allergen or irritant. While it is quite easy to give a textbook description of the typical features of eczema, its clinical manifestations in daily practice are diverse and frequently uncharacteristic. Contact reactions may present as lichenoid, lymphomatoid, granulomatous, pigmented, purpuric, and erythema multiforme-like lesions, thus lacking the typical eczematous appearance and broadening the spectrum of differential diagnoses to be considered. Moreover, a considerable number of agents, acting by mechanisms other than contact, may trigger adverse reactions mimicking the eczematic morphology and distribution pattern of contact dermatitis. Increasingly used in oncology, this is especially true for drugs such as kinase inhibitors. Knowledge of these associations is crucial for the adequate diagnostic and therapeutic care of patients with supposed or actual contact dermatitis.

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“…Primarily hematogenously acting contact allergens include nickel sulfate, chromium compounds, parabens, balsam of Peru, and a variety of pharmaceutical agents .…”

Section: Differential Diagnoses Of Contact Dermatitismentioning

confidence: 99%

Contact dermatitis: relevant differential diagnoses, simulators, and variants

Kränke

Schuster²

2015

J Deutsche Derma Gesell

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“…Patients with previous contact sensitization to certain contact allergens may also develop SAD by ingestion of the same or a crossreacting molecule in food or food additives [5,69]. Spices and other flavorings in patients with contact sensitivity to balsam of Peru (Myroxylon pereirae) [70], cashew nuts containing an allergen that crossreacts with urushiols in poison ivy-sensitive patients, plants such as laurel, chamomile or lettuce containing sesquiterpene lactones, garlic tablets, the antioxidant butylated hydroxyanisole, as well as the preservative sorbic acid and the emulsifier propylene glycol, aluminum in toothpaste and formaldehyde derived from aspartame may cause SAD in sensitized persons [5].…”

Section: Food Allergensmentioning

confidence: 99%

Current understanding of Baboon syndrome

Özkaya

2009

Expert Review of Dermatology

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“…Additional diagnostic testing may be warranted by disease chronicity and severity. Before performing such tests, a positive patch test should be obtained [7]. In step‐wise diagnosis, the next step is a four‐week elimination diet.…”

Section: Special Considerations In Diagnosing Food Allergy In Hematogmentioning

confidence: 99%

“…If the condition has not worsened the challenge should be repeated on day 2. Only if there is an uncertainclinical reactionis a double‐blind, placebo‐controlled re‐provocation recommended[7].…”

Section: Special Considerations In Diagnosing Food Allergy In Hematogmentioning

confidence: 99%

Food allergy

Werfel¹

2008

J Deutsche Derma Gesell

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Food allergy is defined by a specific sensitization against food allergens which is associated with a clinical reaction. Immediate reactions are most common and the skin is most often involved in food allergy. Most food allergies are IgE-mediated although eczema reactions in atopic dermatitis and in hematogenous contact dermatitis to foods can be mediated by specific T-lymphocytes. Only few foods are responsible for the majority of most reactions in childhood. In adults up to 60% of all food allergic reactions are due to cross reactions between foods and inhalative allergens. A stepwise procedure which takes individual factors into account is necessary in the diagnostics of food allergy. The diagnostic algorithm in food allergy is not significantly different from that of other allergies. The oral provocation is the only method to prove food allergy in patients without a convincing history. A specific elimination diet is the only intervention which has been proven to be effective. Further therapeutic approaches are still under study and include specific immunotherapy, specific oral tolerance induction and treatment with anti-IgE antibodies.

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Hämatogene Kontaktekzeme durch Nahrungsmittel

Cited by 20 publications

References 38 publications

Contact dermatitis: relevant differential diagnoses, simulators, and variants

Contact dermatitis: relevant differential diagnoses, simulators, and variants

Current understanding of Baboon syndrome

Food allergy

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