H<scp>istopathological</scp> S<scp>tudies on the</scp> M<scp>echanism of the</scp> F<scp>ormation of</scp> L<scp>eprous</scp> F<scp>oam</scp> C<scp>ells</scp>*

Fukushi, Katsunari

doi:10.1111/j.1440-1827.1959.tb00322.x

Acta Pathologica Japonica

1959

DOI: 10.1111/j.1440-1827.1959.tb00322.x

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Histopathological Studies on the Mechanism of the Formation of Leprous Foam Cells*

Katsunari Fukushi¹

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1969

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A Histopathological Study on the Effect of Various Drugs in Experimental Murine Leprosy

Sasaki¹

1969

Acta Pathologica Japonica

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The effect of a high molecular unsaturated fatty acid C22H36O2 (RTB) In the treatment of experimental murine leprosy was studied for a one‐year long term in parallel with Isonicotinic acid hydrazide (INAH), its derivatives Isonicotinoyl 3–4‐diethoxy benzol hydrazone (No. 284), and a salivary gland hormone (Parotin). The effectiveness of RTB on experimental murileproma was found to be weaker than INAH and No. 284, but almost the same as Parotin. The healing mechanism of RTB and Parotin is not a direct bacteriocidal action, but has a phagocytic and bacteriolytic mechanism of histiocytes due to activation of the reticuloendothelial system. The characteristic findings following RTB administration were predominant proliferation of “argyrophilic fiber type of histiocytes” and argyrophilic fibers, and the new formation of blood‐capillaries. At the same time, the healing process of various drugs and the non‐inhibitory effect of RTB were histopathologically discussed.

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A Histopathological Study on the Effect of Various Drugs in Experimental Murine Leprosy

Sasaki¹

1969

Acta Pathologica Japonica

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Histopathological Studies on the Mechanism of the Formation of Leprous Foam Cells*

Cited by 1 publication

References 14 publications

A Histopathological Study on the Effect of Various Drugs in Experimental Murine Leprosy

A Histopathological Study on the Effect of Various Drugs in Experimental Murine Leprosy

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