Glaucomatouslike visual field defects in chronic papilledema

Grehn, Franz; Knorr-Held, S; Kommerell, Guntram

doi:10.1007/bf00418984

Cited by 14 publications

(4 citation statements)

References 24 publications

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“…With protracted and severe papilledema, nerve fiber layer visual field defects occur. The typical visual field defects found in papilledema are related to nerve fiber bundle damage at the level of the optic disk 13. The papillomacular bundle and thus central visual acuity appears spared until later stages of the disease 14…”

Section: Mechanism and Pattern Of Visual Lossmentioning

confidence: 99%

Papilledema: epidemiology, etiology, and clinical management

Rigi¹,

Almarzouqi

Morgan

et al. 2015

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Papilledema is optic disc swelling due to high intracranial pressure. Possible conditions causing high intracranial pressure and papilledema include intracerebral mass lesions, cerebral hemorrhage, head trauma, meningitis, hydrocephalus, spinal cord lesions, impairment of cerebral sinus drainage, anomalies of the cranium, and idiopathic intracranial hypertension (IIH). Irrespective of the cause, visual loss is the feared morbidity of papilledema, and the main mechanism of optic nerve damage is intraneuronal ischemia secondary to axoplasmic flow stasis. Treatment is directed at correcting the underlying cause. In cases where there is no other identifiable cause for intracranial hypertension (ie, IIH) the available options include both medical and surgical modalities. Weight loss and diuretics remain the mainstays for treatment of IIH, and surgery is typically reserved for patients who fail, are intolerant to, or non-compliant with maximum medical therapy.

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Section: Mechanism and Pattern Of Visual Lossmentioning

confidence: 99%

Papilledema: epidemiology, etiology, and clinical management

Rigi¹,

Almarzouqi

Morgan

et al. 2015

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“…[16] The visual field defects that result from papilledema in IIH are “disc-related defects” and are similar to those found in glaucoma. [17] Visual field losses may be identified in as many as 96% of patients using disease-specific perimetric strategies such as the Armaly-Drance strategy for Goldmann perimetry or SAP. The most common defects seen in IIH are blind spot enlargement, generalized constriction, and loss of the nasal visual fields, especially inferonasal [Fig.…”

Section: Papilledema and Iihmentioning

confidence: 99%

Visual fields in neuro-ophthalmology

Kedar

Ghate

Corbett

2011

Indian J Ophthalmol

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Visual field assessment is important in the evaluation of lesions involving the visual pathways and should be performed at baseline and periodically in the follow-up. Standard automated perimetry has been shown to be adequate in neuro-ophthalmic practise and is now the technique of choice for a majority of practitioners. Goldman kinetic visual fields are useful for patients with severe visual and neurologic deficits and patients with peripheral visual field defects. Visual fields are useful in monitoring progression or recurrence of disease and guide treatment for conditions such as idiopathic intracranial hypertension (IIH), optic neuropathy from multiple sclerosis, pituitary adenomas, and other sellar lesions. They are used as screening tools for toxic optic neuropathy from medications such as ethambutol and vigabatrin. Visual field defects can adversely affect activities of daily living such as personal hygiene, reading, and driving and should be taken into consideration when planning rehabilitation strategies. Visual field testing must be performed in all patients with lesions of the visual pathway.

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“…If papilledema persists over an extended period, irreversible impairment in vision/visual field may occur [ 16 , 17 ]. In this case, approximately a year had passed since the first examination until the patient was diagnosed and treated.…”

Section: Discussionmentioning

confidence: 99%

A Case of Bilateral Papilledema With Improved Clinical Symptoms by Venous Stenting for Superior Sagittal Sinus Stenosis

Miyoshi,

Tabuchi,

Mimura

et al. 2023

Cureus

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Superior sagittal sinus (SSS) obstruction causes intracranial hypertension, often requiring surgical stenting. Consensus on treating brain venous sinus stenosis, another cause, is lacking. This study reports a case of SSS stenosis and intracranial hypertension treated with venous stenting, improving bilateral papilledema. A 51-year-old with a headache and visual disturbance had papilledema and visual field loss. MR venography showed SSS stenosis, leading to a neurosurgery referral. Lumbar puncture confirmed intracranial hypertension (>35 cmH2O), prompting venous stenting. Post-procedure, papilledema, headache, and visual field loss improved. Venous stenting could be effective for SSS stenosis with clinically proven or recurrent pressure differences. Further cases are needed for standardization.

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Glaucomatouslike visual field defects in chronic papilledema

Cited by 14 publications

References 24 publications

Papilledema: epidemiology, etiology, and clinical management

Papilledema: epidemiology, etiology, and clinical management

Visual fields in neuro-ophthalmology

A Case of Bilateral Papilledema With Improved Clinical Symptoms by Venous Stenting for Superior Sagittal Sinus Stenosis

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