2022
DOI: 10.3390/app12031641
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Gingival Recessions and Periodontal Status after Minimum 2-Year-Retention Post-Non-Extraction Orthodontic Treatment

Abstract: The objectives of this study were to assess gingival recessions (GR) and periodontal status in patients previously treated with non-extraction orthodontic treatment and retention at a follow-up of a minimum of two years after the end of treatment. Data from patients aged between 16 and 35 years with a previous non-extraction orthodontic treatment and at least 2 years of retention and full records before and after treatment were collected. The casts were digitalized using the 3Shape TRIOS® intraoral scanner and… Show more

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Cited by 5 publications
(3 citation statements)
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“…Periodontitis is a chronic inflammatory disease associated with the presence of microorganisms and sustained by host-mediated immune-inflammatory response [ 1 , 2 , 3 , 4 ], consequently establishing itself within periodontal tissues and causing their destruction with clinical attachment loss and bone loss [ 5 , 6 , 7 , 8 ] until tooth loss [ 9 ]. The inflammation of periodontal tissues, along with the dysbiotic phenomena of the periodontal microbiome [ 10 , 11 , 12 , 13 ], would also appear to play a role in the pathogenesis of systemic conditions and disorders of degenerative inflammatory and neoplastic nature [ 14 , 15 , 16 , 17 , 18 , 19 ], which, in turn, could influence the onset and, more importantly, the progression of periodontitis. The rate of progression of periodontitis is primarily estimated through both direct clinical and radiographic evidence of periodontal destruction assessed over time and indirect evidence related to biofilm accumulation.…”
Section: Introductionmentioning
confidence: 99%
“…Periodontitis is a chronic inflammatory disease associated with the presence of microorganisms and sustained by host-mediated immune-inflammatory response [ 1 , 2 , 3 , 4 ], consequently establishing itself within periodontal tissues and causing their destruction with clinical attachment loss and bone loss [ 5 , 6 , 7 , 8 ] until tooth loss [ 9 ]. The inflammation of periodontal tissues, along with the dysbiotic phenomena of the periodontal microbiome [ 10 , 11 , 12 , 13 ], would also appear to play a role in the pathogenesis of systemic conditions and disorders of degenerative inflammatory and neoplastic nature [ 14 , 15 , 16 , 17 , 18 , 19 ], which, in turn, could influence the onset and, more importantly, the progression of periodontitis. The rate of progression of periodontitis is primarily estimated through both direct clinical and radiographic evidence of periodontal destruction assessed over time and indirect evidence related to biofilm accumulation.…”
Section: Introductionmentioning
confidence: 99%
“…with systemic diseases [13,14]. In the case of periodontitis, an oral dysbiotic disease [15][16][17][18], suspected periodontopathogens could induce, locally and systemically, a chronic inflammatory state, stimulating the immune response and inflammatory transcription factors, including Nuclear Factor K-beta [19][20][21]. In fact, immune-inflammatory dysregulation is considered the primary key mechanism underlying the potential association of periodontitis with multiple systemic disorders [19][20][21].…”
mentioning
confidence: 99%
“…A slight increase in the risk of developing buccal RBC was detected only in correlation with the presence of a fixed retainer and thin gingival phenotype, mainly in patients with gingivitis. Thus, this research will be extremely useful for clinicians in order to schedule periodical periodontal follow-up appointments, mainly in patients after orthodontic treatment, in order to motivate them to maintain better oral hygiene in adulthood [11].…”
mentioning
confidence: 99%