Genetic variation in lake erie Great Blue Herons (Ardea herodias)

Allozyme polymorphism and interspecific relationships in the Common starling (Sturnus vulgaris) and Spotless starling (S. unicolor) (Aves: Sturnidae)

“…In the Sturnus individuals three zones of activity have been found with one clear band (locus Gtdh-1, Gtdh-3 and Gtdh-4) and one zone with two variants (locus Gtdh-2), as shown in Table 2. The GTDH has been widely used in biochemical systematic of birds (Guttman et al 1980;Ross 1983: Randi et al 1992. (LDH).…”

Genetic variation and prevalence of blood parasites do not correlate among bird species

“…In other words, avian taxa incurring higher prevalence of haematozoans than their sister taxa do not also display consistently higher levels of genetic variation. Evans (1987); 2, Moen (1991); 3, Rhodes, Smith & Chesser (1993); 4, ; 5, Ankney et al (1986); 6, Rhodes, Smith & Chesser (1995); 7, Rhodes, Smith & Smith (1996);8, van Wagner & Baker (1986); 9, Cooke, Parkin & Rockwell (1988); 10, Guttman, Grau & Karlin (1980);11, Baker & Strauch (1986); 12, Gavin, Howard & May (1991); 13, Avise, Patton & Aquadro (1980c); 14, Avise, Patton & Aquadro (1980a); 15, Barrowclough (1980); 16, Barrowclough & Corbin (1978); 17, Zink & Watt (1987); 18, Zink (1982); 19, Barrowclough et al (1985); 20, Zink (1988); 21, Corbin & Wilkie (1988);22, Baker (1976); 23, Corbin (1981); 24, Marten & Johnson (1986); 25, Randi & Spina (1987); 26, Burson (1990); 27, Avise, Patton & Aquadro (1980b); 28, Avise, Aquadro & Patton (1982); 29, Gutierrez, Zink & Yang (1983); 30, Zink, Lott & Anderson (1987); 31, Braun & Robbins (1986); 32, Braun, Kitto & Braun (1984); 33, Stangel, Rodgers & Bryan (1990); 34, Bates & Zink (1992); 35, Parkin & Cole (1984);36, St Louis & Barlow (1988); 37, Randi et al (1992); 38, Gyllensten, Reuterall & Ryman (1979); 39,…”

Genetic variation and prevalence of blood parasites do not correlate among bird species

“…In other words, avian taxa incurring higher prevalence of haematozoans than their sister taxa do not also display consistently higher levels of genetic variation. a H, heterozygosity; P99, polymorphism with criterion that the frequency of the most common allele is 0.99; P95, polymorphism with criterion that the frequency of the most common allele is 0.95. b Sources of the genetic data: 1, Evans (1987); 2, Moen (1991); 3, Rhodes, Smith & Chesser (1993); 4, ; 5, Ankney et al (1986); 6, Rhodes, Smith & Chesser (1995); 7, Rhodes, Smith & Smith (1996);8, van Wagner & Baker (1986); 9, Cooke, Parkin & Rockwell (1988); 10, Guttman, Grau & Karlin (1980);11, Baker & Strauch (1986); 12, Gavin, Howard & May (1991); 13, Avise, Patton & Aquadro (1980c); 14, Avise, Patton & Aquadro (1980a); 15, Barrowclough (1980); 16, Barrowclough & Corbin (1978); 17, Zink & Watt (1987);18, Zink (1982); 19, Barrowclough et al (1985); 20, Zink (1988); 21, Corbin & Wilkie (1988); 22, Baker (1976); 23, Corbin (1981); 24, Marten & Johnson (1986); 25, Randi & Spina (1987); 26, Burson (1990); 27, Avise, Patton & Aquadro (1980b); 28, Avise, Aquadro & Patton (1982); 29, Gutierrez, Zink & Yang (1983); 30, Zink, Lott & Anderson (1987); 31, Braun & Robbins (1986); 32, Braun, Kitto & Braun (1984); 33, Stangel, Rodgers & Bryan (1990); 34, Bates & Zink (1992); 35, Parkin & Cole (1984);36, St Louis & Barlow (1988); 37,…”