Gaze-Evoked Amaurosis

Orcutt, James C.; Tucker, William M.; Mills, Richard P.; Smith, Craig H.

doi:10.1016/s0161-6420(87)33471-2

Cited by 45 publications

(5 citation statements)

References 2 publications

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“…39,40 Gazeevoked amaurosis in papilledema, like orbital tumors, is caused by compression of the short postciliary artery branches and the circle of Zinn-Haller within the peripapillary sclera and or conduction block. 41,42 The deformations in AION and normals demonstrates the effects of eye movement on the optic nerve and loadbearing structures without the confounding effects of elevated CSF fluid pressure. The component of shape deformation that might be influenced by the presence of disc edema is reflected in the difference between AION and normals.…”

Section: Discussionmentioning

confidence: 99%

Gaze Evoked Deformations of the Peripapillary Retina in Papilledema and Ischemic Optic Neuropathy

Sibony

2016

Invest. Ophthalmol. Vis. Sci.

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Horizontal eye movements affect the shape of the ppBM layer. The deformation in normals and AION, in adduction, causes posterior displacement temporal to the BMO. In contrast, the deformations in papilledema are larger, involving temporal and nasal sides, presumably because of shifts in cerebrospinal fluid pressure against the scleral flange and hydraulic stiffening of the optic nerve sheath. The clinical importance of gaze-induced deformations is unknown but repetitive motion may be a factor in the genesis or progression of a variety of optic neuropathies.

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Section: Discussionmentioning

confidence: 99%

Gaze Evoked Deformations of the Peripapillary Retina in Papilledema and Ischemic Optic Neuropathy

Sibony

2016

Invest. Ophthalmol. Vis. Sci.

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“…29 " 31 Therefore, in a given patient with TMVL a variety of causes and mechanisms need to be considered. Because certain TMVL patterns might be caused by specific mechanisms, we evaluated 100 consecutive patients to determine if any TMVL patterns predicted the associated vascular abnormalities.…”

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confidence: 99%

Transient monocular visual loss patterns and associated vascular abnormalities.

Bruno

Corbett

Biller

et al. 1990

Stroke

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“…This manner of transient vision loss is highly suggestive of orbital disease. Orbital meningiomas, optic nerve gliomas, cavernous hemangiomas, orbital osteoma, and orbital fractures are reported to cause gaze-evoked amaurosis [22,Class III]. The proposed mechanisms of vision loss include direct compression of the optic nerve with interference of axonal conduction, or compression of the vascular supply to the optic nerve leading to transient ischemia.…”

Section: Ocular and Nonischemic Causes Of Transient Vision Lossmentioning

confidence: 99%

Transient vision loss

Glazer-Hockstein

Volpe²

2004

Curr Treat Options Neurol

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Transient vision loss often presents a diagnostic dilemma and may occur in many different settings. Episodic vision loss is a common complaint and the physician must distinguish whether the transient vision loss is benign or representative of a more serious underlying condition. Common causes of transient vision loss include ischemia and primary ocular pathology. Ischemic causes of vision loss include retinal vascular disease, intermittent optic nerve vascular compromise, or cerebrovascular disease. Many pathologic ocular processes may lead to transient vision loss, including tear film abnormalities and corneal diseases. The authors discuss the clinical presentation and management of transient vision loss, with special attention on characteristics that differentiate vascular disease from primary ocular pathology.

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Gaze-Evoked Amaurosis

Cited by 45 publications

References 2 publications

Gaze Evoked Deformations of the Peripapillary Retina in Papilledema and Ischemic Optic Neuropathy

Gaze Evoked Deformations of the Peripapillary Retina in Papilledema and Ischemic Optic Neuropathy

Transient monocular visual loss patterns and associated vascular abnormalities.

Transient vision loss

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