2007
DOI: 10.1007/s00423-007-0216-z
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Gastric necrosis complicating lately a Nissen fundoplication

Abstract: Early diagnosis and treatment of gastric dilatation after Nissen fundoplication are essential to prevent from severe secondary complications but can be difficult to establish because of atypical symptoms.

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Cited by 16 publications
(9 citation statements)
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“…4 It is characterized by abdominal discomfort, early satiety, distension, flatulence, and weight loss (secondary to satiety) due to the inability to vomit or belch, resulting from a too narrow fundoplication, increasing the risk of acute gastric dilation. 2,5,6 In this case, the symptoms of gas bloat syndrome were exacerbated by the underlying intestinal occlusion. Two weeks before the current illness, the patient underwent a floppy Nissen fundoplication, involving the division of short vessels and shortening the length of the fundoplication.…”
Section: Discussionmentioning
confidence: 72%
See 1 more Smart Citation
“…4 It is characterized by abdominal discomfort, early satiety, distension, flatulence, and weight loss (secondary to satiety) due to the inability to vomit or belch, resulting from a too narrow fundoplication, increasing the risk of acute gastric dilation. 2,5,6 In this case, the symptoms of gas bloat syndrome were exacerbated by the underlying intestinal occlusion. Two weeks before the current illness, the patient underwent a floppy Nissen fundoplication, involving the division of short vessels and shortening the length of the fundoplication.…”
Section: Discussionmentioning
confidence: 72%
“…Symptoms include difficulty belching and vomiting, early satiety, distension, flatulence, and weight loss due to early satiety. 2 In a patient with intestinal obstruction and a history of fundoplication, acute gastric dilation may occur due to the inability to vomit, creating a vicious circle where an increase in intragastric pressure enhances lower esophageal sphincter incompetence, resulting in increased gastric pressure due to the inability to vomit 1 . Persistent elevation of intragastric pressure causes vascular compromise of the gastric wall.…”
Section: Introductionmentioning
confidence: 99%
“…Unlike previous studies, Dynamic MRI was used for the first time to evaluate gastric fundus and splenic circulation in patients who underwent LNF, which was performed dissecting the GB. The development of novel MRI may provide a new opportunity for evaluating abdominal pathologies [ 13 ].…”
Section: Discussionmentioning
confidence: 99%
“…Some studies have shown that an intragastric pressure greater than 20-30 cm H2O is necessary to cause the occlusion of the gastric luminal blood circulation [15] . Cases of gastric necrosis following acute small bowel obstruction based on adhesions and gastric outlet obstruction based on antral gastric cancer and trichophytobezoar after an antireflux technique [3][4][5] have been reported. In infants, this rare post Nissen complication has also been described [16] .…”
Section: Discussionmentioning
confidence: 99%
“…gastricae breves). Notwithstanding this fact, acute gastric dilatation accompanied with or without gastric outlet obstruction [2][3][4][5][6][7] , eating disorders [8][9][10] or gas-bloat syndrome are recognized causes of ischemic gastric necrosis.…”
Section: Introductionmentioning
confidence: 99%