Gastric Function in Pregnancy

Hunt, J. N.; Murray, Fiona

doi:10.1111/j.1471-0528.1958.tb06212.x

Cited by 71 publications

(15 citation statements)

References 8 publications

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“…Generally, though, no delay in gastric emptying has been shown with pregnancy (Davison et al 1970;Hunt & Murray, 1978). Certainly the small intestine is affected by pregnancy.…”

Section: Decreased Luteal Phase Gut Motilitymentioning

confidence: 99%

Bowel Function, Food Intake and the Menstrual Cycle

Vlitos

Davies

1996

Nutr. Res. Rev.

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“…Generally, though, no delay in gastric emptying has been shown with pregnancy (Davison et al 1970;Hunt & Murray, 1978). Certainly the small intestine is affected by pregnancy.…”

Section: Decreased Luteal Phase Gut Motilitymentioning

confidence: 99%

Bowel Function, Food Intake and the Menstrual Cycle

Vlitos

Davies

1996

Nutr. Res. Rev.

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“…The results demonstrated that a reduction of pepsin secretion in pregnancy, when the concentration of ovarian hormones is high [64], a decrease in pepsin secretion induced by ovarian hormones in rats [65], inhibition of pepsin secretion in ovariectomized (OVX) rats by estradiol [66], and an increase in pepsin secretion followed by gastric mucosal inflammation after stress [67].…”

Section: Tbi Gi Tract and Sex Hormonesmentioning

confidence: 99%

Traumatic Brain Injury and the Gastrointestinal Tract: The Role of Female Sexual Hormones

et al. 2017

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Traumatic brain injury (TBI) is an important public health problem which can lead to several complications like gastrointestinal dysfunction. Patients with TBI often suffer from gastrointestinal dysfunctions such as enteral feeding intolerance. Gastric ulceration is induced by various forms of stress like surgery, ischemia and trauma. TBI causes a delayed but significant decrease in intestinal contractile activity in the ileum leading to delayed transport. The association between severity of brain injury and enteral feeding intolerance suggests a strong link between the central nervous system and the non-functional gut. The results of recent basic and clinical studies revealed the beneficial effects of estrogen and progesterone hormones in the treatment of gastrointestinal dysfunction. Females have more resistance to stress and fewer gastrointestinal lesions occur in females as compared to males. Sex steroid hormones have sparked interest as possible neuroprotective agents after traumatic injury and many studies have been done on this subject. This article reviews the literature related to some possible physiological effects of female sexual hormones on the gastrointestinal tract following TBI.

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“…First, the growth of the mammary glands during lactation provides a new compartment of drug distribution, excretion (Clewell and Gearhart, 2002), and possibly metabolism as alcohol dehydrogenase (ADH) is expressed in breast tissue (Saleem et al, 1984;Triano et al, 2003). Second, the high nutritional demands associated with milk production coincide with remarkable changes in the gastrointestinal tract (Hammond, 1997;Hunt and Murray, 1958;Uvnas-Moberg et al, 1987). In rodents, lactation is associated with dramatic intestinal growth (e.g., increased height of the villi) and a generalized hypertrophy and hyperplasia of the mucosal epithelium (Hammond, 1997).…”

mentioning

confidence: 99%

“…Given the physiological adaptations of the digestive system during lactation (Hammond, 1997;Hunt and Murray, 1958;Uvnas-Moberg et al, 1987), the present study tested the hypotheses that lactation alters alcohol pharmacokinetics and that the metabolic differences between lactating and nonlactating women would be more striking when analyzing alcohol pharmacokinetics when alcohol was consumed with a meal. To this end, we studied lactating women who were exclusively breast feeding a 2-to 5-month-old infant and 2 control groups of nonlactating women under 2 conditions: when alcohol was consumed following a meal or on an empty stomach.…”

mentioning

confidence: 99%