Gall-stone ileus

Stitt, R. B.; Heslin, Donna; Currie, Donald J.

doi:10.1002/bjs.1800540803

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2005

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Cited by 9 publications

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“…This clinical entity has been reported at a rate of 4.7% [3]. Carefully searching for more stones throughout the intestinal tract is important [3] because multiple stones can be expected in 3-44% of all patients with gallstone ileus [3,21,[25][26][27][28]. In our experience, three cases (7.5%) of recurrent disease were noted in patients who previ-…”

Section: Discussionmentioning

confidence: 83%

Role of Helical CT in Diagnosis of Gallstone Ileus and Related Conditions

Lassandro

Romano

Ragozzino

et al. 2005

American Journal of Roentgenology

130

105

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Section: Discussionmentioning

confidence: 83%

Role of Helical CT in Diagnosis of Gallstone Ileus and Related Conditions

Lassandro

Romano

Ragozzino

et al. 2005

American Journal of Roentgenology

130

105

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“…1 Gall stone ileus occurs when a gall stone erodes into part of the gastrointestinal system and causes intestinal obstruction. .…”

mentioning

confidence: 99%

Gastric outflow obstruction caused by gall stones and leading to death by complex metabolic derangement.

Wight

Seed²,

Yeo³

et al. 1997

J Clin Pathol

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A 67 year old woman was admitted with a three week history of vomiting, having become increasingly confused for three days. Investigations revealed deranged serum biochemistry consistent with a combination of a diabetic non-ketotic hyperosmolar state and a metabolic alkalosis consistent with gastric outflow obstruction. She was treated with intravenous saline, intravenous insulin, and subcutaneous heparin, but did not improve clinically and had an asystolic cardiac arrest the following day; she was transferred to the intensive care unit and despite treatment with inotropes she died 40 hours after admission. Necropsy revealed that the stomach was massively dilated with gas and stomach contents, and contained many small black faceted gall stones. In addition a large nonfaceted brown-yellow gall stone was wedged in the pyloric antrum causing total obstruction. The patient had died from a complex metabolic derangement including non-ketotic hyperosmotic diabetic coma and metabolic alkalosis precipitated by the acute gastric outflow obstruction complicated by previously undiagnosed type II diabetes mellitus.

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