1991
DOI: 10.1016/0014-4886(91)90024-7
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GABAergic neurotransmission in the C57BL10, spssps mouse mutant: A model of absence seizures

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Cited by 10 publications
(1 citation statement)
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“…The results of BZD-GABAA receptor binding in animal models of absence seizures are very dependent on the model studied. In the tottering mouse, [3H]flunitrazepam binding to the BZD-GABAA receptor was normal (Psarropoulou et al, 1987), but in a mouse mutant with audiogenic "absences" [3H]flunitrazepam binding was reduced in all areas except hippocampus (Ortiz et al, 1991). In inbred rats with spontaneous absence seizures, which are believed to be more representative of the condition in humans, [3H]flunitrazepam binding was normal (Knight and Bowery, 1992;Snead et al, 1992).…”
Section: Discussionmentioning
confidence: 99%
“…The results of BZD-GABAA receptor binding in animal models of absence seizures are very dependent on the model studied. In the tottering mouse, [3H]flunitrazepam binding to the BZD-GABAA receptor was normal (Psarropoulou et al, 1987), but in a mouse mutant with audiogenic "absences" [3H]flunitrazepam binding was reduced in all areas except hippocampus (Ortiz et al, 1991). In inbred rats with spontaneous absence seizures, which are believed to be more representative of the condition in humans, [3H]flunitrazepam binding was normal (Knight and Bowery, 1992;Snead et al, 1992).…”
Section: Discussionmentioning
confidence: 99%