Further Investigations on the Pathophysiology of the Compartmental Syndrome

Sheridan, Geoffrey W.; Matsen, F A; Krugmire, Richard B.

doi:10.1097/00003086-197703000-00070

Cited by 34 publications

(19 citation statements)

References 0 publications

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“…Simple direct nerve stimulation, using an inexpensive battery-powered stimulator and two 25-gauge needles, may help to differentiate compartment syndrome from a proximal nerve injury [40]. The myoneural junction is most sensitive to ischaemia [41] and therefore muscles paralysed because of compartmental syndrome would not respond to nerve stimulation.…”

Section: Discussionmentioning

confidence: 99%

Acute compartment syndrome secondary to diabetic muscle infarction: case report and literature review

Woolley

Smith

2006

European Journal of Emergency Medicine

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Acute compartment syndrome has a multitude of aetiologies. Unfortunately, the diagnosis is often delayed, resulting in permanent functional loss. Although spontaneous muscle infarction is an uncommon, yet well-recognized complication of diabetes mellitus, subsequent development of compartment syndrome appears to be rare, with only five case reports identified in the literature. This condition has not been reported in the emergency medicine literature. We report a case of a diabetic gentleman who presented with lower limb pain significantly out of proportion to any obvious injury and had a subsequent diagnosis of acute compartment syndrome. Despite fasciotomies, he had a persistent foot drop. Nontraumatic acute compartment syndrome secondary to diabetic muscle infarction should be considered in any diabetic patient presenting with pain out of proportion to sustained injury.

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Section: Discussionmentioning

confidence: 99%

Acute compartment syndrome secondary to diabetic muscle infarction: case report and literature review

Woolley

Smith

2006

European Journal of Emergency Medicine

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“…Skin fold chambers, arterial occlusion via fogerty balloon, arterial ligation, inflation of latex balloons within compartments, external compression and tourniquet application are some of the techniques published. [32][33][34][35][36][37][38][39][40] Large animal canine models deemed clinically relevant have induced CS using pressurecontrolled autologous blood or plasma infusion into compartments.…”

Section: Compartment Syndrome Modellingmentioning

confidence: 99%

“…33 The experimental time of 45 minutes was applied to observe the early microvascular response to EICP and its subsequent effects on the surrounding tissue. Small animal models are not identical to metabolic and cellular derangements in humans, and, hence, experimental effects need to be compared with the existing body of literature.…”

Section: Compartment Syndrome Modellingmentioning

confidence: 99%

Compartment syndrome–induced microvascular dysfunction: an experimental rodent model

Lawendy

Sanders²,

Bihari³

et al. 2011

Can J Surg

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“…If not treated promptly, the pressure reaches critical levels resulting in ischemia of the contained tissues, principally muscle, nerve, or bone. [1][2][3][4][5][6] Potential sequelae include infection, muscle necrosis, neurologic deficits, contractures, fracture nonunion, chronic pain, and in the most severe cases amputation or death. [7][8][9][10][11][12] It has been acknowledged for more than 40 years that the single cause of a poor outcome in ACS is delay in diagnosis.…”

Section: Introductionmentioning

confidence: 99%