1999
DOI: 10.1016/s0024-3205(99)90521-0
|View full text |Cite
|
Sign up to set email alerts
|

Functional and molecular identification and characterization of cardiac M3 and M4 muscarinic acetylcholine receptors

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2

Citation Types

0
2
0

Year Published

1999
1999
1999
1999

Publication Types

Select...
1

Relationship

0
1

Authors

Journals

citations
Cited by 1 publication
(2 citation statements)
references
References 0 publications
0
2
0
Order By: Relevance
“…Recently, the existence of M 3 receptors in canine atrial myocytes has been convincingly demonstrated with both functional and molecular evidence in our laboratory. We found that the cardiac M 3 receptors are functionally coupled to a novel type of delayed rectifier K + channels ( Shi et al ., 1998a , 1998b ) and we named this K + current I KM3 (M 3 receptor‐mediated K + current). We have confirmed that the cardiac M 3 receptors and I KM3 can be activated by tetramethylammonium ( Shi et al ., 1998b ) and choline ( Shi et al ., 1998c ).…”
Section: Introductionmentioning
confidence: 99%
See 1 more Smart Citation
“…Recently, the existence of M 3 receptors in canine atrial myocytes has been convincingly demonstrated with both functional and molecular evidence in our laboratory. We found that the cardiac M 3 receptors are functionally coupled to a novel type of delayed rectifier K + channels ( Shi et al ., 1998a , 1998b ) and we named this K + current I KM3 (M 3 receptor‐mediated K + current). We have confirmed that the cardiac M 3 receptors and I KM3 can be activated by tetramethylammonium ( Shi et al ., 1998b ) and choline ( Shi et al ., 1998c ).…”
Section: Introductionmentioning
confidence: 99%
“…We found that the cardiac M 3 receptors are functionally coupled to a novel type of delayed rectifier K + channels ( Shi et al ., 1998a , 1998b ) and we named this K + current I KM3 (M 3 receptor‐mediated K + current). We have confirmed that the cardiac M 3 receptors and I KM3 can be activated by tetramethylammonium ( Shi et al ., 1998b ) and choline ( Shi et al ., 1998c ). Preliminary data have been obtained in our laboratory indicating that pilocarpine can also activate a K + current with properties almost identical to TMA‐ or choline‐induced currents in dog atrial cells and the stimulation of mAChRs, likely the M 3 receptor subtype, is required for the current activation ( Wang et al ., 1998 ).…”
Section: Introductionmentioning
confidence: 99%