2015
DOI: 10.1136/bcr-2014-208603
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Fulminant crural compartment syndrome preceded by psychogenic polydipsia

Abstract: SUMMARYWe report a case of bilateral anterolateral crural compartment syndrome elicited by hyponatraemia and psychogenic polydipsia. The unusual constellation of clinical findings and diminished pain expression made initial diagnostic procedures challenging. The possible pathogenesis and treatment options are discussed. Impairment of lower extremity function at follow-up was serious and permanent. BACKGROUND

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Cited by 6 publications
(5 citation statements)
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References 21 publications
(15 reference statements)
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“…Compartment syndrome isolated to the anterior compartment and from a medically induced aetiology is very rare. Our review found only five published cases [1][2][3][4][5]. Four cases were secondary to psychogenic polydipsia, and one case attributed to severe primary hypothyroidism and adrenal insufficiency.…”
Section: Discussionmentioning
confidence: 92%
See 1 more Smart Citation
“…Compartment syndrome isolated to the anterior compartment and from a medically induced aetiology is very rare. Our review found only five published cases [1][2][3][4][5]. Four cases were secondary to psychogenic polydipsia, and one case attributed to severe primary hypothyroidism and adrenal insufficiency.…”
Section: Discussionmentioning
confidence: 92%
“…All of the published cases similar to the one described in this case report resulted in permanent disability due to delays in recognising the diagnosis. Drop foot and loss of sensation in the dorsal foot is the likely outcome for a patient with delayed release of the anterior compartment [1,3,4]. This is due to necrosis of the muscles in the anterior compartment and/or compression of the deep peroneal nerve as it passes through the compartment, supplying the muscles before giving sensation to the 1st webspace on the dorsum of the foot.…”
Section: Discussionmentioning
confidence: 99%
“…The underlying pathophysiology of this condition has not been elucidated, but one hypothesis is that increased intracellular calcium followed by decreased ion exchange could lead to muscle cell death. Another theory is that fluctuations in extracellular osmolarity could alter transmembrane potentials, resulting in muscle cell death [ 6 ]. Although the causes of water-intoxication-related rhabdomyolysis seem multifactorial, rapid correction of serum sodium has been proposed as a risk factor in several studies [ 10 , 11 ].…”
Section: Discussionmentioning
confidence: 99%
“…Psychogenic polydipsia though commonly found with patients of schizophrenia can be a potentially fatal condition. There have been reports of life threatening conditions such as seizure, rhabdomyolysis, aspiration pneumonia, and crural compartment syndrome associated with psychogenic polydipsia [79]. There are also reports of death secondary to self-induced water intoxication in patients with schizophrenia [10].…”
Section: Discussionmentioning
confidence: 99%