Fully covered removable nitinol self-expandable metal stents (SEMS) in malignant strictures of the esophagus: a multicenter analysis

Abstract

Our study suggests that fully covered SEMS placement improve dysphagia scores in patients with malignant strictures, particularly in the unresectable population. Further technical improvements in design to minimize long-term malfunction and migration are required.

Use of covered self-expandable stents for benign colorectal disorders in children

“…Traditionally this technique is frequently used in adults for palliation of malignant dysphagia [11] and intestinal obstruction caused by colorectal cancer [1,3]. With regard to the pediatric population stenting for benign esophageal conditions seems to become a promising tool particularly for anastomotic strictures and leaks post esophageal atresia repair and perforations after dilatation [10,12,13].…”

A bridging stent to surgery in patients with esophageal and gastroesophageal junction cancer has a dramatic negative impact on patient survival

“…Several studies have demonstrated that stenting malignant strictures improves dysphagia and nutritional status. 10,13,14 Several studies have evaluated the safety and efficacy of stenting esophageal cancer as treatment for dysphagia during neoadjuvant therapy. [14][15][16][17][18][19][20][21][22][23][24] However, no studies to date have investigated the oncological impact of stenting as a bridge to surgery without neoadjuvant treatment.…”

Use of Fully Covered Self-Expandable Metal Stents for Benign Esophageal Disorders in Children

“…SEMSs are collapsed and inserted via a delivery system, which facilitates placement, minimizes the need for dilation, and reduces the incidence of trauma and perforation during insertion. 1 Benign indications in which SEMSs have been used more or less successfully in adults are iatrogenic and spontaneous esophageal perforations, tracheoesophageal fistulas, and anastomotic leaks, as well as strictures from caustic and idiopathic causes. 2,3 Children may develop esophageal sequelae from a variety of causes, including ingestion of corrosives, gastroesophageal reflux (GER), eosinophilic esophagitis, drug injuries, sclerotherapy, radiation, fibrotic healing after surgical repair of esophageal atresia (EA), idiopathic conditions, and iatrogenic injuries.…”