Focal hepatic vein stenoses in diffuse liver disease.

Lorenz, Jonathan M.; Winsberg, Fred

doi:10.7863/jum.1996.15.4.313

Cited by 6 publications

(4 citation statements)

References 2 publications

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“…associatedwith liver diseasemay be the result of increased resistance in the hepaticvein cir culation caused by focal stenoses [20]. A pro found change in hepatic vein pulsatility was also found during pregnancy.…”

Section: Dietrich Et Almentioning

confidence: 71%

Hepatic and portal vein flow pattern in correlation with intrahepatic fat deposition and liver histology in patients with chronic hepatitis C.

Dietrich

Lee²,

Gottschalk³

et al. 1998

American Journal of Roentgenology

101

112

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OBJECTIVE.The flow pattern in hepatic veins depends on cardiac physiology and liver histology. The aim of our study was to determine the dependence of the flow pattern of he patic and portal veins in relation to histologic features in patients with chronic hepatitis C. SUBJECTS AND METHODS.In 135 patients with chronic hepatitis C, the Doppler sonography spectrum of the right hepatic vein was classified as triphasic, biphasic, or monophasic. The flow of theportalvein wascharacterizedaccordingto theundulation(veloc itYm@min). A liver biopsy was performed during sonography, and biopsy specimens were semiquantitatively evaluatedon a histologicactivity index anda scoreof the hepaticfat con tent. Multiple logistic regression analysis was used to identify the histologic features that might contribute to the type of flow pattern. RESULTS.The hepatocyte fat content was the only variable associated with an indepen dent effect on the type of flow pattern (monophasic versus triphasic; odds ratio, 16.26; 95% confidence interval, 6.38â€"41.45; p < .0001). A pronounced undulation in the portal vein was associated with portal inflammation but not with other parameters of the histologic activity in dex or the intrahepatic fat deposition. The normal triphasic Doppler sonography waveformof hepaticveinsvanesaccordingto changes in the central venous pressure. During atrial and ventricular diastole, two portions of the signal (phase I and phase II) reflect the hepatofugalflow to the heart, while during atrial contraction a third portion (phase ifi) representsa short interval of reversedflow [1,10]. Conflicting data have been reported concerning changes of the flow pattern in rela tion to chronic liver disease. A few studiessug gested that liver fibrosis or cirrhosis and transplant rejection may reduce undulation of the hepatic flow pattern [2][3][4]7]. The possible contribution of inflammatory changes and mtra hepaticfat depositionhasneverbeensystemati cally investigated.To address thesequestions, chronichepatitis C is an attractiveclinical model [11â€"13] be causethe diseasecan causethe completehisto logic spectrum of inflammatory and fibrotic changes[14â€"17] andcan lead to variable intra hepaticfat deposition [3,18]. In the present study, we examined 135 patients with chronic hepatitis C and 75 age-and sex-matched healthy subjectsusing B-mode and duplex

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Section: Dietrich Et Almentioning

confidence: 71%

Hepatic and portal vein flow pattern in correlation with intrahepatic fat deposition and liver histology in patients with chronic hepatitis C.

Dietrich

Lee²,

Gottschalk³

et al. 1998

American Journal of Roentgenology

101

112

View full text Add to dashboard Cite

show abstract

“…8 In line with this, a higher incidence of esophageal varices has been reported in cirrhotic patients with a flat hepatic vein waveform. 18 In another study, a damped waveform in hepatic veins was associated with focal hepatic vein stenoses, which can occur in diffuse liver disease 17,19 and in metastatic liver disease. 20 Other groups reported a damped Doppler waveform in hepatic veins in patients with Budd-Chiari syndrome.…”

Section: Discussionmentioning

confidence: 99%

Association between duplex Doppler sonographic flow pattern in right hepatic vein and various liver diseases

Herbay

Frieling

Häussinger

2001

J. Clin. Ultrasound

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“…22 The mechanism for this loss of hepatic vein pulsatility is unclear, but is likely related to hepatic vein stenosis, which is caused by impression on the hepatic veins by regenerating nodules. 23 Although measurement of vessel diameters and velocities and calculation of various indices have shown promise in certain studies, the most reliable and widely used approach for the diagnosis of portal hypertension is the detection of portal systemic collaterals. In general, portal systemic collaterals can be divided into tributary collaterals and developed collaterals.…”

Section: Portal Hypertensionmentioning

confidence: 99%

Doppler Sonography of Portal Hypertension

Robinson¹,

Middleton²,

Al-Sukaiti³

et al. 2009

Ultrasound Quarterly

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Doppler sonography is frequently obtained in patients with chronic liver disease, patients with suspected portal hypertension, and patients with known portal hypertension. In this clinical setting, it is important to understand both the normal hemodynamics of the liver and the morphological and hemodynamic changes that occur with portal hypertension. Among the many findings of portal hypertension are enlargement of the portal vein, decreased portal vein velocity, increased congestion index, development of portosystemic collaterals, and reversal of portal vein flow. The most important portal systemic collaterals to focus on with Doppler are the coronary vein and the umbilical vein. Using all of these clues, it is possible to diagnose portal hypertension with a high degree of sensitivity and specificity in most patients.

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Focal hepatic vein stenoses in diffuse liver disease.

Cited by 6 publications

References 2 publications

Hepatic and portal vein flow pattern in correlation with intrahepatic fat deposition and liver histology in patients with chronic hepatitis C.

Hepatic and portal vein flow pattern in correlation with intrahepatic fat deposition and liver histology in patients with chronic hepatitis C.

Association between duplex Doppler sonographic flow pattern in right hepatic vein and various liver diseases

Doppler Sonography of Portal Hypertension

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